rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
11
|
pubmed:dateCreated |
2008-11-10
|
pubmed:abstractText |
Understanding of the molecular pathophysiology of spondylarthritis (SpA) remains largely elusive. This is related both to the complexity of the disease (axial versus peripheral disease, inflammation versus tissue remodeling) and to the difficulty in obtaining samples from primary disease sites. This study was undertaken to explore a gene expression approach for identifying novel candidate mediators of SpA.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
AIM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Nov
|
pubmed:issn |
0004-3591
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:volume |
58
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
3430-5
|
pubmed:meshHeading |
pubmed-meshheading:18975340-Adult,
pubmed-meshheading:18975340-Arthritis, Rheumatoid,
pubmed-meshheading:18975340-Enzyme-Linked Immunosorbent Assay,
pubmed-meshheading:18975340-Female,
pubmed-meshheading:18975340-Gene Expression,
pubmed-meshheading:18975340-Humans,
pubmed-meshheading:18975340-Interleukin-7,
pubmed-meshheading:18975340-Male,
pubmed-meshheading:18975340-Microarray Analysis,
pubmed-meshheading:18975340-Multigene Family,
pubmed-meshheading:18975340-RNA, Messenger,
pubmed-meshheading:18975340-Reverse Transcriptase Polymerase Chain Reaction,
pubmed-meshheading:18975340-Spondylarthritis,
pubmed-meshheading:18975340-Synovitis
|
pubmed:year |
2008
|
pubmed:articleTitle |
Identification of interleukin-7 as a candidate disease mediator in spondylarthritis.
|
pubmed:affiliation |
Hannover Medical School, Hannover, Germany. Rihl.Markus@MH-Hannover.de
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|