Linked Life Data

18954925

Source:http://linkedlifedata.com/resource/pubmed/id/18954925

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
5
pubmed:dateCreated
2009-4-6
pubmed:abstractText
Plants react to O(3) threat by setting up a variety of defensive strategies involving the co-ordinated modulation of stress perception, signalling and metabolic responses. Although stomata largely controls O(3) uptake, differences in O(3) tolerance cannot always be ascribed to changes in stomatal conductance but cell protective and repair processes should be taken into account. O(3)-driven ROS production in the apoplast induces a secondary, active, self-propagating generation of ROS, whose levels must be finely tuned, by many enzymatic and non-enzymatic antioxidant systems, to induce gene activation without determining uncontrolled cell death. Additional signalling molecules, as ethylene, jasmonic and salicylic acid are also crucial to determine the spreading and the containment of leaf lesions. The main recent results obtained on O(3) sensing, signal transduction, ROS formation and detoxification mechanisms are here discussed.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
May
pubmed:issn
1873-6424
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
157
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1461-9
pubmed:meshHeading
pubmed:year
2009
pubmed:articleTitle
Detoxification and repair process of ozone injury: from O3 uptake to gene expression adjustment.
pubmed:affiliation
Department of Agricultural Chemistry and Biotechnology, University of Pisa, Via del Borghetto 80, 56124 Pisa, Italy. castagna@agr.unipi.it
pubmed:publicationType
Journal Article, Review