pubmed-article:18853744 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C0227525 | lld:lifeskim |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C0010805 | lld:lifeskim |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C0071277 | lld:lifeskim |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C0699900 | lld:lifeskim |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C0243125 | lld:lifeskim |
pubmed-article:18853744 | lifeskim:mentions | umls-concept:C1709059 | lld:lifeskim |
pubmed-article:18853744 | pubmed:issue | 5 | lld:pubmed |
pubmed-article:18853744 | pubmed:dateCreated | 2008-10-15 | lld:pubmed |
pubmed-article:18853744 | pubmed:abstractText | Hepatoma cells are morphologically different from those of the normal liver. Intermediate filaments (IFs) are important in building the cellular architecture and maintaining the outline of cells. Plectin is a cross-linking protein that organizes the cytoskeleton into a stable meshwork, which can maintain the uniform size and shape of hepatocytes. Apoptosis might be the most possible pathway for creating plectin deficiency in the in vivo state. | lld:pubmed |
pubmed-article:18853744 | pubmed:language | eng | lld:pubmed |
pubmed-article:18853744 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18853744 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:18853744 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18853744 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18853744 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18853744 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18853744 | pubmed:issn | 0258-851X | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:YehKun-TuKT | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:HsuYung-Hsian... | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:PeiRen-JengRJ | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:ChaoWei-TingW... | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:LiuYi-HsiangY... | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:ChengChiung-C... | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:HoChin-ChinCC | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:LaiYih-Shyong... | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:TsaiMing-Chua... | lld:pubmed |
pubmed-article:18853744 | pubmed:author | pubmed-author:HoLu-ChangLC | lld:pubmed |
pubmed-article:18853744 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:18853744 | pubmed:volume | 22 | lld:pubmed |
pubmed-article:18853744 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18853744 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18853744 | pubmed:pagination | 543-8 | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:meshHeading | pubmed-meshheading:18853744... | lld:pubmed |
pubmed-article:18853744 | pubmed:articleTitle | Degradation of plectin with modulation of cytokeratin 18 in human liver cells during staurosporine-induced apoptosis. | lld:pubmed |
pubmed-article:18853744 | pubmed:affiliation | Department of Pathology, Jen Ai Hospital, Taichung, Taiwan, R.O.C. | lld:pubmed |
pubmed-article:18853744 | pubmed:publicationType | Journal Article | lld:pubmed |