pubmed-article:18838388 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C0003873 | lld:lifeskim |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C0920317 | lld:lifeskim |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C0021755 | lld:lifeskim |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C0205419 | lld:lifeskim |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C0033413 | lld:lifeskim |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C0004083 | lld:lifeskim |
pubmed-article:18838388 | lifeskim:mentions | umls-concept:C1882417 | lld:lifeskim |
pubmed-article:18838388 | pubmed:issue | 12 | lld:pubmed |
pubmed-article:18838388 | pubmed:dateCreated | 2008-11-12 | lld:pubmed |
pubmed-article:18838388 | pubmed:abstractText | IL-1 has a central role mediating inflammation and joint destruction in RA. Single nucleotide polymorphisms (SNPs) and haplotype structure in the promoter region can modulate IL-1 function. This study examined the effects of four common promoter SNPs in the IL-1 region on susceptibility and clinical characteristics of RA in British Caucasian patients and assessed the risk of RA by meta-analysis of published studies. | lld:pubmed |
pubmed-article:18838388 | pubmed:commentsCorrections | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18838388 | pubmed:language | eng | lld:pubmed |
pubmed-article:18838388 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18838388 | pubmed:citationSubset | AIM | lld:pubmed |
pubmed-article:18838388 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18838388 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18838388 | pubmed:month | Dec | lld:pubmed |
pubmed-article:18838388 | pubmed:issn | 1462-0332 | lld:pubmed |
pubmed-article:18838388 | pubmed:author | pubmed-author:PointonJ JJJ | lld:pubmed |
pubmed-article:18838388 | pubmed:author | pubmed-author:HarrisonPP | lld:pubmed |
pubmed-article:18838388 | pubmed:author | pubmed-author:ChapmanKK | lld:pubmed |
pubmed-article:18838388 | pubmed:author | pubmed-author:WordsworthB... | lld:pubmed |
pubmed-article:18838388 | pubmed:author | pubmed-author:RoddamAA | lld:pubmed |
pubmed-article:18838388 | pubmed:issnType | Electronic | lld:pubmed |
pubmed-article:18838388 | pubmed:volume | 47 | lld:pubmed |
pubmed-article:18838388 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18838388 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18838388 | pubmed:pagination | 1768-70 | lld:pubmed |
pubmed-article:18838388 | pubmed:dateRevised | 2011-7-1 | lld:pubmed |
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pubmed-article:18838388 | pubmed:meshHeading | pubmed-meshheading:18838388... | lld:pubmed |
pubmed-article:18838388 | pubmed:meshHeading | pubmed-meshheading:18838388... | lld:pubmed |
pubmed-article:18838388 | pubmed:meshHeading | pubmed-meshheading:18838388... | lld:pubmed |
pubmed-article:18838388 | pubmed:meshHeading | pubmed-meshheading:18838388... | lld:pubmed |
pubmed-article:18838388 | pubmed:meshHeading | pubmed-meshheading:18838388... | lld:pubmed |
pubmed-article:18838388 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18838388 | pubmed:articleTitle | Interleukin-1 promoter region polymorphism role in rheumatoid arthritis: a meta-analysis of IL-1B-511A/G variant reveals association with rheumatoid arthritis. | lld:pubmed |
pubmed-article:18838388 | pubmed:affiliation | Botnar Research Centre, Nuffield Orthopaedic Centre, Windmill Road, Oxford OX3 7LD, UK. pille.harrison@ndos.ox.ac.uk | lld:pubmed |
pubmed-article:18838388 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18838388 | pubmed:publicationType | Review | lld:pubmed |
pubmed-article:18838388 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
pubmed-article:18838388 | pubmed:publicationType | Meta-Analysis | lld:pubmed |
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