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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2008-12-16
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pubmed:abstractText |
Human atherosclerotic plaques express markers of macrophage/dendritic cells as well as high levels of inflammatory proteins such as secreted phospholipase A(2) type IIA (sPLA(2)-IIA). To understand the cellular changes associated with the progress of atherosclerosis, we evaluated the role of sPLA(2)-IIA in mediating monocyte recruitment and differentiation into antigen-presenting cells.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1755-3245
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:day |
1
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pubmed:volume |
81
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
54-63
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pubmed:meshHeading |
pubmed-meshheading:18755682-Animals,
pubmed-meshheading:18755682-Antigens, CD,
pubmed-meshheading:18755682-Antigens, CD14,
pubmed-meshheading:18755682-Atherosclerosis,
pubmed-meshheading:18755682-Cell Adhesion Molecules,
pubmed-meshheading:18755682-Cell Differentiation,
pubmed-meshheading:18755682-Cell Line,
pubmed-meshheading:18755682-Chemotaxis,
pubmed-meshheading:18755682-Dendritic Cells,
pubmed-meshheading:18755682-Dose-Response Relationship, Drug,
pubmed-meshheading:18755682-Endocytosis,
pubmed-meshheading:18755682-Group II Phospholipases A2,
pubmed-meshheading:18755682-Humans,
pubmed-meshheading:18755682-Immunity, Active,
pubmed-meshheading:18755682-Immunity, Innate,
pubmed-meshheading:18755682-Mice,
pubmed-meshheading:18755682-Monocytes,
pubmed-meshheading:18755682-Transfection
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pubmed:year |
2009
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pubmed:articleTitle |
Secreted phospholipase A2 type IIA as a mediator connecting innate and adaptive immunity: new role in atherosclerosis.
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pubmed:affiliation |
Instituto de Biología y Genética Molecular, Consejo Superior de Investigaciones Científicas/Universidad de Valladolid, C/ Sanz y Fores s/n., 47005 Valladolid, Spain.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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