pubmed-article:18576327 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:18576327 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
pubmed-article:18576327 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
pubmed-article:18576327 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
pubmed-article:18576327 | lifeskim:mentions | umls-concept:C0011644 | lld:lifeskim |
pubmed-article:18576327 | lifeskim:mentions | umls-concept:C1419227 | lld:lifeskim |
pubmed-article:18576327 | pubmed:issue | 7 | lld:pubmed |
pubmed-article:18576327 | pubmed:dateCreated | 2008-7-15 | lld:pubmed |
pubmed-article:18576327 | pubmed:abstractText | Activated adhesive signaling is a hallmark of fibroblasts isolated from the scars of scleroderma (systemic sclerosis [SSc]) lesions. Rac1 plays a key role in adhesive signaling. The aim of the present study was to examine the role of Rac1 in bleomycin-induced scleroderma, using mice with a fibroblast-specific deletion of Rac1. | lld:pubmed |
pubmed-article:18576327 | pubmed:grant | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18576327 | pubmed:language | eng | lld:pubmed |
pubmed-article:18576327 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18576327 | pubmed:citationSubset | AIM | lld:pubmed |
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pubmed-article:18576327 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:18576327 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:18576327 | pubmed:month | Jul | lld:pubmed |
pubmed-article:18576327 | pubmed:issn | 0004-3591 | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:LeaskAndrewA | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:GlogauerMicha... | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:AbrahamDavid... | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:DentonChristo... | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:Shi-wenXuX | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:KennedyLauraL | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:KapoorMohitM | lld:pubmed |
pubmed-article:18576327 | pubmed:author | pubmed-author:LiuShangxiS | lld:pubmed |
pubmed-article:18576327 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:18576327 | pubmed:volume | 58 | lld:pubmed |
pubmed-article:18576327 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:18576327 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:18576327 | pubmed:pagination | 2189-95 | lld:pubmed |
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pubmed-article:18576327 | pubmed:meshHeading | pubmed-meshheading:18576327... | lld:pubmed |
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pubmed-article:18576327 | pubmed:meshHeading | pubmed-meshheading:18576327... | lld:pubmed |
pubmed-article:18576327 | pubmed:year | 2008 | lld:pubmed |
pubmed-article:18576327 | pubmed:articleTitle | Role of Rac1 in a bleomycin-induced scleroderma model using fibroblast-specific Rac1-knockout mice. | lld:pubmed |
pubmed-article:18576327 | pubmed:affiliation | University of Western Ontario, London, Ontario, Canada. | lld:pubmed |
pubmed-article:18576327 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:18576327 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:18576327 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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