| pubmed-article:18469846 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:18469846 | lifeskim:mentions | umls-concept:C0012655 | lld:lifeskim |
| pubmed-article:18469846 | lifeskim:mentions | umls-concept:C0030193 | lld:lifeskim |
| pubmed-article:18469846 | lifeskim:mentions | umls-concept:C0206745 | lld:lifeskim |
| pubmed-article:18469846 | lifeskim:mentions | umls-concept:C0521329 | lld:lifeskim |
| pubmed-article:18469846 | lifeskim:mentions | umls-concept:C0441472 | lld:lifeskim |
| pubmed-article:18469846 | lifeskim:mentions | umls-concept:C0205217 | lld:lifeskim |
| pubmed-article:18469846 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:18469846 | pubmed:dateCreated | 2008-6-30 | lld:pubmed |
| pubmed-article:18469846 | pubmed:abstractText | Annexin-A1 (ANXA1), a glucocorticoid-regulated protein, mediates several of the anti-inflammatory actions of the glucocorticoids. Previous studies demonstrated that ANXA1 is involved in pain modulation. The current study, using ANXA1 knockout mice (ANXA1-/-), is aimed at addressing the site and mechanism of the modulatory action of ANXA1 as well as possible involvement of ANXA1 in mediating the analgesic action of glucocorticoids. | lld:pubmed |
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| pubmed-article:18469846 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:18469846 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:18469846 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:18469846 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:18469846 | pubmed:issn | 0007-1188 | lld:pubmed |
| pubmed-article:18469846 | pubmed:author | pubmed-author:FlowerR JRJ | lld:pubmed |
| pubmed-article:18469846 | pubmed:author | pubmed-author:AyoubS SSS | lld:pubmed |
| pubmed-article:18469846 | pubmed:author | pubmed-author:YazidSS | lld:pubmed |
| pubmed-article:18469846 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:18469846 | pubmed:volume | 154 | lld:pubmed |
| pubmed-article:18469846 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:18469846 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:18469846 | pubmed:pagination | 1135-42 | lld:pubmed |
| pubmed-article:18469846 | pubmed:dateRevised | 2009-11-18 | lld:pubmed |
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| pubmed-article:18469846 | pubmed:year | 2008 | lld:pubmed |
| pubmed-article:18469846 | pubmed:articleTitle | Increased susceptibility of annexin-A1 null mice to nociceptive pain is indicative of a spinal antinociceptive action of annexin-A1. | lld:pubmed |
| pubmed-article:18469846 | pubmed:affiliation | Centre for Biochemical Pharmacology, William Harvey Research Institute, St Bart's and the London School of Medicine and Dentistry, Queen Mary, University of London, Charterhouse Square, London, UK. s.s.ayoub@qmul.ac.uk | lld:pubmed |
| pubmed-article:18469846 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:18469846 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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