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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
5
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pubmed:dateCreated |
2008-4-21
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pubmed:abstractText |
Several recent studies have shown that a brief ischaemia applied during the onset of reperfusion (postconditioning) is cardioprotective in different animal models. The potential application of postconditioning to organs different from the heart, i.e. kidney, is not available and is investigated in the present study. We also tested the hypothesis that mitochondria play a central role in renal protection during reperfusion.
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pubmed:commentsCorrections |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
May
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pubmed:issn |
1460-2385
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
23
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1504-12
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pubmed:dateRevised |
2009-6-23
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pubmed:meshHeading |
pubmed-meshheading:18285396-Animals,
pubmed-meshheading:18285396-Blood Urea Nitrogen,
pubmed-meshheading:18285396-Creatinine,
pubmed-meshheading:18285396-Electron Transport,
pubmed-meshheading:18285396-Hydrogen Peroxide,
pubmed-meshheading:18285396-Ischemic Preconditioning,
pubmed-meshheading:18285396-Kidney,
pubmed-meshheading:18285396-Male,
pubmed-meshheading:18285396-Mitochondria,
pubmed-meshheading:18285396-Oxidative Stress,
pubmed-meshheading:18285396-Proteins,
pubmed-meshheading:18285396-Rats,
pubmed-meshheading:18285396-Rats, Wistar,
pubmed-meshheading:18285396-Reperfusion Injury,
pubmed-meshheading:18285396-Time Factors
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pubmed:year |
2008
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pubmed:articleTitle |
Postconditioning is an effective strategy to reduce renal ischaemia/reperfusion injury.
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pubmed:affiliation |
Division of Nephrology, Department of Biomedical Sciences, University of Foggia Viale Pinto, 71100 Foggia, Italy.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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