pubmed-article:17965172 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C0242726 | lld:lifeskim |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C1095831 | lld:lifeskim |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C0004755 | lld:lifeskim |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C0007603 | lld:lifeskim |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C0596902 | lld:lifeskim |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C0019868 | lld:lifeskim |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C2587213 | lld:lifeskim |
pubmed-article:17965172 | lifeskim:mentions | umls-concept:C0597484 | lld:lifeskim |
pubmed-article:17965172 | pubmed:issue | 4 | lld:pubmed |
pubmed-article:17965172 | pubmed:dateCreated | 2007-12-6 | lld:pubmed |
pubmed-article:17965172 | pubmed:abstractText | Plant salinity tolerance is a polygenic trait with contributions from genetic, developmental, and physiological interactions, in addition to interactions between the plant and its environment. In this study, we show that in salt-tolerant genotypes of barley (Hordeum vulgare), multiple mechanisms are well combined to withstand saline conditions. These mechanisms include: (1) better control of membrane voltage so retaining a more negative membrane potential; (2) intrinsically higher H(+) pump activity; (3) better ability of root cells to pump Na(+) from the cytosol to the external medium; and (4) higher sensitivity to supplemental Ca(2+). At the same time, no significant difference was found between contrasting cultivars in their unidirectional (22)Na(+) influx or in the density and voltage dependence of depolarization-activated outward-rectifying K(+) channels. Overall, our results are consistent with the idea of the cytosolic K(+)-to-Na(+) ratio being a key determinant of plant salinity tolerance, and suggest multiple pathways of controlling that important feature in salt-tolerant plants. | lld:pubmed |
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pubmed-article:17965172 | pubmed:language | eng | lld:pubmed |
pubmed-article:17965172 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:17965172 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:17965172 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:17965172 | pubmed:month | Dec | lld:pubmed |
pubmed-article:17965172 | pubmed:issn | 0032-0889 | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:TesterMarkM | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:ShabalaSergey... | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:PalmgrenMicha... | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:PottosinIgor... | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:ChenZhonghuaZ | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:CuinTracey... | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:FuglsangAnja... | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:NewmanIan AIA | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:ZhouMeixueM | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:JhaDeepaD | lld:pubmed |
pubmed-article:17965172 | pubmed:author | pubmed-author:Zepeda-JazoIs... | lld:pubmed |
pubmed-article:17965172 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:17965172 | pubmed:volume | 145 | lld:pubmed |
pubmed-article:17965172 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:17965172 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:17965172 | pubmed:pagination | 1714-25 | lld:pubmed |
pubmed-article:17965172 | pubmed:dateRevised | 2010-9-16 | lld:pubmed |
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