|
rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0001511,
umls-concept:C0007634,
umls-concept:C0014175,
umls-concept:C0015350,
umls-concept:C0026336,
umls-concept:C0043210,
umls-concept:C0205217,
umls-concept:C0227843,
umls-concept:C0334094,
umls-concept:C0449258,
umls-concept:C0449432,
umls-concept:C0598002,
umls-concept:C0871261,
umls-concept:C1179435,
umls-concept:C1516240,
umls-concept:C1524073,
umls-concept:C1548799,
umls-concept:C1704632,
umls-concept:C1705248,
umls-concept:C1706817,
umls-concept:C2911692
|
|
pubmed:issue |
12
|
|
pubmed:dateCreated |
2007-11-20
|
|
pubmed:abstractText |
Endometriosis, classified as the presence of endometrial cells in ectopic sites, is a debilitating disease causing pain and infertility in approximately 10% of women of reproductive age. It is associated with the aberrant expression of extracellular matrix (ECM) components and their receptors, integrins.
|
|
pubmed:grant |
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Dec
|
|
pubmed:issn |
0268-1161
|
|
pubmed:author |
|
|
pubmed:issnType |
Print
|
|
pubmed:volume |
22
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
3139-47
|
|
pubmed:meshHeading |
pubmed-meshheading:17921481-Adult,
pubmed-meshheading:17921481-Cell Adhesion,
pubmed-meshheading:17921481-Cell Division,
pubmed-meshheading:17921481-Cell Line,
pubmed-meshheading:17921481-Collagen Type I,
pubmed-meshheading:17921481-Collagen Type IV,
pubmed-meshheading:17921481-DNA,
pubmed-meshheading:17921481-Disease Progression,
pubmed-meshheading:17921481-Endometriosis,
pubmed-meshheading:17921481-Endometrium,
pubmed-meshheading:17921481-Extracellular Matrix Proteins,
pubmed-meshheading:17921481-Female,
pubmed-meshheading:17921481-Humans,
pubmed-meshheading:17921481-Integrins,
pubmed-meshheading:17921481-Laminin,
pubmed-meshheading:17921481-Middle Aged,
pubmed-meshheading:17921481-Stromal Cells
|
|
pubmed:year |
2007
|
|
pubmed:articleTitle |
Endometrial cells from women with endometriosis have increased adhesion and proliferative capacity in response to extracellular matrix components: towards a mechanistic model for endometriosis progression.
|
|
pubmed:affiliation |
Nuffield Department of Obstetrics and Gynaecology, Level 3, The Women's Centre, John Radcliffe Hospital, Oxford OX3 9DU, UK.
|
|
pubmed:publicationType |
Journal Article,
In Vitro,
Research Support, Non-U.S. Gov't
|
