| pubmed-article:17703315 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C0009324 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C1171362 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C0812445 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C1332821 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C1707170 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C0017262 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C1515670 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C2709058 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C0547047 | lld:lifeskim |
| pubmed-article:17703315 | lifeskim:mentions | umls-concept:C0304604 | lld:lifeskim |
| pubmed-article:17703315 | pubmed:issue | 12 | lld:pubmed |
| pubmed-article:17703315 | pubmed:dateCreated | 2007-10-5 | lld:pubmed |
| pubmed-article:17703315 | pubmed:abstractText | Ulcerative colitis is characterized by relapsing mucosal inflammation where the lesions include tissue-damaging granulocytes. In addition, T cells and natural killer (NK) cells play important pathophysiologic roles. Chemokines are a large family of peptides that play key roles in the regulation of inflammation. The CXC-chemokines, growth-related oncogene (GRO)-alpha/CXCL1 and interleukin (IL)-8/CXCL8, both recruit neutrophils and possess mitogenic properties, whereas the interferon-dependent CXC-chemokines monokine induced by gamma-interferon (MIG)/CXCL9, interferon-gamma inducible protein of 10 kD/CXCL10, and IFN-inducible T cell alpha chemoattractant/CXCL11 recruit and activate T cells and NK cells. | lld:pubmed |
| pubmed-article:17703315 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:17703315 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:17703315 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:17703315 | pubmed:month | Dec | lld:pubmed |
| pubmed-article:17703315 | pubmed:issn | 0179-1958 | lld:pubmed |
| pubmed-article:17703315 | pubmed:author | pubmed-author:EgestenArneA | lld:pubmed |
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| pubmed-article:17703315 | pubmed:author | pubmed-author:SangfeltPerP | lld:pubmed |
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| pubmed-article:17703315 | pubmed:author | pubmed-author:EliassonMette... | lld:pubmed |
| pubmed-article:17703315 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:17703315 | pubmed:volume | 22 | lld:pubmed |
| pubmed-article:17703315 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:17703315 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:17703315 | pubmed:pagination | 1421-7 | lld:pubmed |
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| pubmed-article:17703315 | pubmed:year | 2007 | lld:pubmed |
| pubmed-article:17703315 | pubmed:articleTitle | The proinflammatory CXC-chemokines GRO-alpha/CXCL1 and MIG/CXCL9 are concomitantly expressed in ulcerative colitis and decrease during treatment with topical corticosteroids. | lld:pubmed |
| pubmed-article:17703315 | pubmed:affiliation | Department of Clinical Sciences Lund, BMC B14, Tornavagen 10, 221 84, Lund, Sweden. Arne.Egesten@med.lu.se | lld:pubmed |
| pubmed-article:17703315 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:17703315 | pubmed:publicationType | Controlled Clinical Trial | lld:pubmed |
| pubmed-article:17703315 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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