Source:http://linkedlifedata.com/resource/pubmed/id/17695812
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
5
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pubmed:dateCreated |
2007-8-16
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pubmed:abstractText |
The complement system plays an important role in renal pathogenesis, and C5b-9, a terminal complement complex, is regarded as the principal mediator of proteinuria in idiopathic membranous nephropathy(MN). Since factor H regulates complement activation at the C3 step and is a crucial factor in complement-mediated tissue injury, the urinary excretion of factor H in patients with idiopathic MN was investigated.
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pubmed:language |
jpn
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:issn |
0385-2385
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
49
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
499-504
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pubmed:dateRevised |
2011-8-1
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pubmed:meshHeading |
pubmed-meshheading:17695812-Adult,
pubmed-meshheading:17695812-Biological Markers,
pubmed-meshheading:17695812-Complement Factor H,
pubmed-meshheading:17695812-Complement Membrane Attack Complex,
pubmed-meshheading:17695812-Female,
pubmed-meshheading:17695812-Glomerulonephritis, Membranous,
pubmed-meshheading:17695812-Humans,
pubmed-meshheading:17695812-Male,
pubmed-meshheading:17695812-Middle Aged,
pubmed-meshheading:17695812-Proteinuria
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pubmed:year |
2007
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pubmed:articleTitle |
[Evaluation of urinary factor H excretion in patients with idiopathic membranous nephropathy].
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pubmed:affiliation |
Faculty of Human Health Science, Hachinohe University, Aomori, Japan.
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pubmed:publicationType |
Journal Article,
English Abstract
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