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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
8
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pubmed:dateCreated |
2007-7-19
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pubmed:abstractText |
Colon cancer is the third most prevalent cancer in the United States. However, the molecular mechanisms involved in the development and progression of colon cancer are incompletely understood. This study was initiated to explore the potential role of the receptor for advanced glycation end-products and S100P in modulation of key properties of human colon cancer cells.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
0012-3706
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
50
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
1230-40
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pubmed:dateRevised |
2007-12-3
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pubmed:meshHeading |
pubmed-meshheading:17587138-Adenocarcinoma,
pubmed-meshheading:17587138-Calcium-Binding Proteins,
pubmed-meshheading:17587138-Cell Culture Techniques,
pubmed-meshheading:17587138-Cell Line, Tumor,
pubmed-meshheading:17587138-Cell Movement,
pubmed-meshheading:17587138-Cell Proliferation,
pubmed-meshheading:17587138-Colonic Neoplasms,
pubmed-meshheading:17587138-Humans,
pubmed-meshheading:17587138-MAP Kinase Signaling System,
pubmed-meshheading:17587138-Neoplasm Proteins,
pubmed-meshheading:17587138-Phosphorylation,
pubmed-meshheading:17587138-Receptors, Immunologic
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pubmed:year |
2007
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pubmed:articleTitle |
RAGE activation by S100P in colon cancer stimulates growth, migration, and cell signaling pathways.
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pubmed:affiliation |
Program in Cellular & Molecular Biology, University of Michigan Medical School, Ann Arbor, MI 48109-0622, USA.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't,
Research Support, N.I.H., Extramural
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