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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
8
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pubmed:dateCreated |
2007-8-13
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pubmed:abstractText |
Oxidative stress and myocyte apoptosis are thought to play an important role in the pathogenesis, progression and prognosis of heart failure (HF). Heat shock protein 27 (Hsp27) has been found to confer resistance to oxidative stress in cultured cells; however, the role of Hsp27 in in-vivo hearts remains to be determined.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Aug
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pubmed:issn |
1388-9842
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
9
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
762-9
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pubmed:dateRevised |
2011-6-8
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pubmed:meshHeading |
pubmed-meshheading:17481944-Animals,
pubmed-meshheading:17481944-Antibiotics, Antineoplastic,
pubmed-meshheading:17481944-Apoptosis,
pubmed-meshheading:17481944-Blotting, Western,
pubmed-meshheading:17481944-Disease Models, Animal,
pubmed-meshheading:17481944-Doxorubicin,
pubmed-meshheading:17481944-HSP27 Heat-Shock Proteins,
pubmed-meshheading:17481944-Heart Failure,
pubmed-meshheading:17481944-Heat-Shock Proteins,
pubmed-meshheading:17481944-Immunohistochemistry,
pubmed-meshheading:17481944-In Situ Nick-End Labeling,
pubmed-meshheading:17481944-Mice,
pubmed-meshheading:17481944-Mice, Transgenic,
pubmed-meshheading:17481944-Mitochondria, Heart,
pubmed-meshheading:17481944-Myocytes, Cardiac,
pubmed-meshheading:17481944-Organ Size,
pubmed-meshheading:17481944-Oxidative Stress,
pubmed-meshheading:17481944-Reactive Oxygen Species
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pubmed:year |
2007
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pubmed:articleTitle |
Over-expression of heat shock protein 27 attenuates doxorubicin-induced cardiac dysfunction in mice.
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pubmed:affiliation |
Department of Geriatrics, First Affiliated Hospital with Nanjing Medical University, Nanjing, China.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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