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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2007-7-30
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pubmed:abstractText |
Type 2 diabetes mellitus shows a characteristic altered platelet function that can be due to several mechanisms such as oxidative stress. Hyperhomocysteinemia, considered as a risk factor for various arterial thrombosis, may have a role in generating oxidative damage, even if the pathogenic mechanisms are still not clear. In this report we aimed to determine the role of plasma homocysteine in inducing oxidative stress in type 2 diabetes mellitus.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
0049-3848
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
120
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
607-13
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pubmed:meshHeading |
pubmed-meshheading:17188741-Aged,
pubmed-meshheading:17188741-Case-Control Studies,
pubmed-meshheading:17188741-Cyclic GMP,
pubmed-meshheading:17188741-Diabetes Mellitus, Type 2,
pubmed-meshheading:17188741-Glutathione,
pubmed-meshheading:17188741-Homocysteine,
pubmed-meshheading:17188741-Humans,
pubmed-meshheading:17188741-Male,
pubmed-meshheading:17188741-Middle Aged,
pubmed-meshheading:17188741-Nitric Oxide,
pubmed-meshheading:17188741-Platelet Activation,
pubmed-meshheading:17188741-Reactive Oxygen Species
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pubmed:year |
2007
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pubmed:articleTitle |
Homocysteine, reactive oxygen species and nitric oxide in type 2 diabetes mellitus.
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pubmed:affiliation |
Department of Experimental Medicine, Biochemistry Section, University of Genoa, Italy.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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