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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
1
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pubmed:dateCreated |
2007-2-6
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pubmed:abstractText |
Contralateral nephrectomy stimulates compensatory growth of the remaining kidney. Intensive growth is frequently associated with increased apoptosis. The proliferation and apoptosis of cultured rat mesangial cells isolated from the remaining kidney following contralateral nephrectomy were evaluated. The involvement of the renin-angiotensin system was concomitantly assessed.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Jan
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pubmed:issn |
1234-1010
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
13
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
BR16-23
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pubmed:dateRevised |
2010-11-18
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pubmed:meshHeading |
pubmed-meshheading:17179902-Angiotensin I,
pubmed-meshheading:17179902-Angiotensin II,
pubmed-meshheading:17179902-Angiotensin II Type 2 Receptor Blockers,
pubmed-meshheading:17179902-Animals,
pubmed-meshheading:17179902-Apoptosis,
pubmed-meshheading:17179902-Bradykinin,
pubmed-meshheading:17179902-Captopril,
pubmed-meshheading:17179902-Cell Proliferation,
pubmed-meshheading:17179902-Cell Separation,
pubmed-meshheading:17179902-Cells, Cultured,
pubmed-meshheading:17179902-Gene Expression Regulation,
pubmed-meshheading:17179902-Mesangial Cells,
pubmed-meshheading:17179902-Nephrectomy,
pubmed-meshheading:17179902-RNA, Messenger,
pubmed-meshheading:17179902-Rats,
pubmed-meshheading:17179902-Rats, Sprague-Dawley,
pubmed-meshheading:17179902-Receptor, Angiotensin, Type 1,
pubmed-meshheading:17179902-Renin-Angiotensin System
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pubmed:year |
2007
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pubmed:articleTitle |
Apoptosis and proliferation of mesangial cells isolated from kidneys undergoing compensatory growth following contralateral nephrectomy: role of the renin-angiotensin system.
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pubmed:affiliation |
Nephrology Division, Assaf Harofeh Medical Center, Affiliated to Sackler Faculty of Medicine, Tel Aviv University, Israel. labnefro@asaf.health.gov.il
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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