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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
6
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pubmed:dateCreated |
2006-12-4
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pubmed:abstractText |
Brain injury after trauma is an important cause of mortality and morbidity in society. There is evidence in both man and laboratory animals that in addition to necrosis, cell loss may occur as a result of programmed cell death (PCD). The cellular and molecular responses after head injury are partly influenced by genetic polymorphisms of apolipoprotein E and the pro-inflammatory cytokine IL-I.
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pubmed:grant |
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:issn |
0722-5091
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
25
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
255-64
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pubmed:dateRevised |
2009-11-19
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pubmed:meshHeading |
pubmed-meshheading:17140155-Adolescent,
pubmed-meshheading:17140155-Adult,
pubmed-meshheading:17140155-Aged,
pubmed-meshheading:17140155-Apolipoprotein E4,
pubmed-meshheading:17140155-Apoptosis,
pubmed-meshheading:17140155-Biological Markers,
pubmed-meshheading:17140155-Brain Injuries,
pubmed-meshheading:17140155-Cell Count,
pubmed-meshheading:17140155-DNA Mutational Analysis,
pubmed-meshheading:17140155-Female,
pubmed-meshheading:17140155-Gene Frequency,
pubmed-meshheading:17140155-Genetic Predisposition to Disease,
pubmed-meshheading:17140155-Genetic Testing,
pubmed-meshheading:17140155-Genotype,
pubmed-meshheading:17140155-Hippocampus,
pubmed-meshheading:17140155-Humans,
pubmed-meshheading:17140155-In Situ Nick-End Labeling,
pubmed-meshheading:17140155-Interleukin-1,
pubmed-meshheading:17140155-Interleukin-1alpha,
pubmed-meshheading:17140155-Interleukin-1beta,
pubmed-meshheading:17140155-Male,
pubmed-meshheading:17140155-Middle Aged,
pubmed-meshheading:17140155-Nerve Degeneration,
pubmed-meshheading:17140155-Polymorphism, Genetic
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pubmed:articleTitle |
No evidence for the presence of apolipoprotein epsilon4, interleukin-lalpha allele 2 and interleukin-1beta allele 2 cause an increase in programmed cell death following traumatic brain injury in humans.
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pubmed:affiliation |
Academic Unit of Neuropathology, University of Glasgow, Glasgow, Scotland, UK.
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pubmed:publicationType |
Journal Article,
Research Support, N.I.H., Extramural
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