| pubmed-article:16918969 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16918969 | lifeskim:mentions | umls-concept:C0024301 | lld:lifeskim |
| pubmed-article:16918969 | lifeskim:mentions | umls-concept:C0376515 | lld:lifeskim |
| pubmed-article:16918969 | lifeskim:mentions | umls-concept:C1517478 | lld:lifeskim |
| pubmed-article:16918969 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
| pubmed-article:16918969 | lifeskim:mentions | umls-concept:C1515021 | lld:lifeskim |
| pubmed-article:16918969 | pubmed:issue | 3 | lld:pubmed |
| pubmed-article:16918969 | pubmed:dateCreated | 2006-8-21 | lld:pubmed |
| pubmed-article:16918969 | pubmed:abstractText | Follicular lymphoma (FL) arising primarily in the skin has recently been proposed as a distinct entity on the basis of a low incidence of t(14;18)(q32;q21) and bcl-2 expression, with a very high percentage of patients surviving more than 5 years. However, cases of t(14;18)(q32;q21)-positive primary cutaneous FL (PCFL) and examples of t(14;18)(q32;q21)-negative FL at nodal and other extranodal sites, are well documented. The aim of this study was to test the hypothesis that there is a subtype of FL lacking t(14;18)(q32;q21), which preferentially involves certain sites but is not restricted by anatomical location. | lld:pubmed |
| pubmed-article:16918969 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16918969 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16918969 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16918969 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16918969 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16918969 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16918969 | pubmed:month | Sep | lld:pubmed |
| pubmed-article:16918969 | pubmed:issn | 0309-0167 | lld:pubmed |
| pubmed-article:16918969 | pubmed:author | pubmed-author:LevisonD ADA | lld:pubmed |
| pubmed-article:16918969 | pubmed:author | pubmed-author:WhiteJ MJM | lld:pubmed |
| pubmed-article:16918969 | pubmed:author | pubmed-author:GoodladJ RJR | lld:pubmed |
| pubmed-article:16918969 | pubmed:author | pubmed-author:KernohanN MNM | lld:pubmed |
| pubmed-article:16918969 | pubmed:author | pubmed-author:HamiltonD ADA | lld:pubmed |
| pubmed-article:16918969 | pubmed:author | pubmed-author:BatstoneP JPJ | lld:pubmed |
| pubmed-article:16918969 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16918969 | pubmed:volume | 49 | lld:pubmed |
| pubmed-article:16918969 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16918969 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16918969 | pubmed:pagination | 229-41 | lld:pubmed |
| pubmed-article:16918969 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:16918969 | pubmed:meshHeading | pubmed-meshheading:16918969... | lld:pubmed |
| pubmed-article:16918969 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16918969 | pubmed:articleTitle | BCL2 gene abnormalities define distinct clinical subsets of follicular lymphoma. | lld:pubmed |
| pubmed-article:16918969 | pubmed:affiliation | Department of Pathology, Western General Hospital, Edinburgh, and Division of Pathology and Neuroscience, University of Dundee, UK. john.goodlad@luht.scot.nhs.uk | lld:pubmed |
| pubmed-article:16918969 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16918969 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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