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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2006-5-2
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pubmed:abstractText |
Neuropeptide Y (NPY) has been shown to modulate seizure activities. To provide further understanding of the involvement of two of the most abundantly expressed NPY receptors, Y1 and Y2, we assessed the effect of Y1 and Y2 gene deletion on systemic kainic acid-induced seizures. We also examined the effect of rAAV-mediated hippocampal NPY overexpression on seizure susceptibility in these receptor knockout mice.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
0013-9580
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pubmed:author |
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pubmed:issnType |
Print
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pubmed:volume |
47
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
773-80
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:16650144-Animals,
pubmed-meshheading:16650144-DNA, Recombinant,
pubmed-meshheading:16650144-Dependovirus,
pubmed-meshheading:16650144-Genetic Vectors,
pubmed-meshheading:16650144-Hippocampus,
pubmed-meshheading:16650144-Kainic Acid,
pubmed-meshheading:16650144-Male,
pubmed-meshheading:16650144-Mice,
pubmed-meshheading:16650144-Mice, Inbred C57BL,
pubmed-meshheading:16650144-Mice, Knockout,
pubmed-meshheading:16650144-Neuropeptide Y,
pubmed-meshheading:16650144-Receptors, Neuropeptide Y,
pubmed-meshheading:16650144-Seizures
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pubmed:year |
2006
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pubmed:articleTitle |
Differential actions of NPY on seizure modulation via Y1 and Y2 receptors: evidence from receptor knockout mice.
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pubmed:affiliation |
Department of Molecular Medicine and Pathology, University of Auckland, Auckland, New Zealand. d.lin@garvan.org.au
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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