Source:http://linkedlifedata.com/resource/pubmed/id/16491297
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
2006-2-21
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pubmed:abstractText |
VC is an important clinical entity; however, very little information is available on its resolution. Induction and regression of calcitriol-induced VC was studied in 47 rats. After calcitriol withdrawal, there was a relatively rapid regression of VC mediated by an active cellular process.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Mar
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pubmed:issn |
0884-0431
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
21
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
484-90
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:16491297-Animals,
pubmed-meshheading:16491297-Aorta,
pubmed-meshheading:16491297-Aortic Diseases,
pubmed-meshheading:16491297-Calcinosis,
pubmed-meshheading:16491297-Calcitriol,
pubmed-meshheading:16491297-Kidney,
pubmed-meshheading:16491297-Male,
pubmed-meshheading:16491297-Rats,
pubmed-meshheading:16491297-Rats, Wistar
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pubmed:year |
2006
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pubmed:articleTitle |
Reversibility of calcitriol-induced medial artery calcification in rats with intact renal function.
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pubmed:affiliation |
Department of Medicina y Cirugia Animal, Universidad de Cordoba, Cordoba, Spain.
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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