Source:http://linkedlifedata.com/resource/pubmed/id/16392220
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
3
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pubmed:dateCreated |
2006-1-5
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pubmed:abstractText |
In conclusion, reduced nitric oxide bioactivity contributes to cardiac and vascular remodeling. Nitricoxide-enhancing therapy can slow cardiac andvascular remodeling. African-Americans with heart failure exhibit nitric oxide deficiency and a dramatic benefit from nitric-oxide-enhancing therapy. The potential benefit of this therapy in other groups with heart failure and in other vascular remodeling disease processes requires further study.
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pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:issn |
0730-2347
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
32
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
366-8
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pubmed:dateRevised |
2008-11-20
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pubmed:meshHeading |
pubmed-meshheading:16392220-African Americans,
pubmed-meshheading:16392220-Drug Therapy, Combination,
pubmed-meshheading:16392220-Endothelium, Vascular,
pubmed-meshheading:16392220-Heart Failure,
pubmed-meshheading:16392220-Humans,
pubmed-meshheading:16392220-Hydralazine,
pubmed-meshheading:16392220-Isosorbide Dinitrate,
pubmed-meshheading:16392220-Treatment Outcome,
pubmed-meshheading:16392220-United States,
pubmed-meshheading:16392220-Vasodilator Agents
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pubmed:year |
2005
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pubmed:articleTitle |
A-HeFT: old dog, new endothelial tricks.
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pubmed:affiliation |
Rasmussen Center for Cardiovascular Disease Prevention, Department of Medicine, University of Minnesota, Minneapolis, Minnesota 55455, USA. cohnx001@umn.edu
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pubmed:publicationType |
Journal Article,
Review
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