Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2005-12-5
pubmed:abstractText
The pathophysiology of HIV-1-related intestinal dysfunction is largely unknown. We previously found that the transactivator factor peptide (Tat) produced by HIV-1 induces ion secretion and inhibits cell proliferation in human enterocytes. Because sugar malabsorption is a frequent feature in AIDS patients, we evaluated whether Tat inhibits intestinal glucose absorption.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jan
pubmed:issn
0269-9370
pubmed:author
pubmed:issnType
Print
pubmed:day
2
pubmed:volume
20
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
5-10
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed-meshheading:16327313-3-Pyridinecarboxylic acid..., pubmed-meshheading:16327313-Biological Markers, pubmed-meshheading:16327313-Blotting, Western, pubmed-meshheading:16327313-Caco-2 Cells, pubmed-meshheading:16327313-Calcium Channel Agonists, pubmed-meshheading:16327313-Calcium Channels, pubmed-meshheading:16327313-Culture Media, pubmed-meshheading:16327313-Cytoskeleton, pubmed-meshheading:16327313-Dose-Response Relationship, Drug, pubmed-meshheading:16327313-Epithelial Cells, pubmed-meshheading:16327313-Fluorescent Antibody Technique, pubmed-meshheading:16327313-Gene Products, tat, pubmed-meshheading:16327313-Glucose, pubmed-meshheading:16327313-HIV-1, pubmed-meshheading:16327313-Humans, pubmed-meshheading:16327313-Intestinal Absorption, pubmed-meshheading:16327313-Sodium-Glucose Transporter 1, pubmed-meshheading:16327313-Tubulin, pubmed-meshheading:16327313-tat Gene Products, Human Immunodeficiency Virus
pubmed:year
2006
pubmed:articleTitle
Inhibitory effect of HIV-1 Tat protein on the sodium-D-glucose symporter of human intestinal epithelial cells.
pubmed:affiliation
Department of Pediatrics, University Federico II of Naples, Italy.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't