Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
21
pubmed:dateCreated
2005-11-2
pubmed:abstractText
Several tumor suppressor pathways have been identified as modulators of telomerase function. We examined the functional role of the retinoblastoma-E2F1 pathway in regulating telomerase activity in malignant gliomas.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Nov
pubmed:issn
1460-2105
pubmed:author
pubmed:issnType
Electronic
pubmed:day
2
pubmed:volume
97
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
1589-600
pubmed:dateRevised
2010-11-18
pubmed:meshHeading
pubmed-meshheading:16264179-Animals, pubmed-meshheading:16264179-Brain Neoplasms, pubmed-meshheading:16264179-Cell Line, Tumor, pubmed-meshheading:16264179-Chromatin Immunoprecipitation, pubmed-meshheading:16264179-DNA-Binding Proteins, pubmed-meshheading:16264179-E2F1 Transcription Factor, pubmed-meshheading:16264179-Gene Expression Regulation, Enzymologic, pubmed-meshheading:16264179-Gene Expression Regulation, Neoplastic, pubmed-meshheading:16264179-Genetic Linkage, pubmed-meshheading:16264179-Glioblastoma, pubmed-meshheading:16264179-Humans, pubmed-meshheading:16264179-Immunoblotting, pubmed-meshheading:16264179-Mice, pubmed-meshheading:16264179-Mice, Transgenic, pubmed-meshheading:16264179-Predictive Value of Tests, pubmed-meshheading:16264179-Prognosis, pubmed-meshheading:16264179-Promoter Regions, Genetic, pubmed-meshheading:16264179-RNA, Messenger, pubmed-meshheading:16264179-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:16264179-Sarcoma, pubmed-meshheading:16264179-Telomerase
pubmed:year
2005
pubmed:articleTitle
Expression of transcription factor E2F1 and telomerase in glioblastomas: mechanistic linkage and prognostic significance.
pubmed:affiliation
Department of Neuro-Oncology, University of Texas M.D. Anderson Cancer Center, Houston, TX 77030, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, N.I.H., Extramural