Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
12
pubmed:dateCreated
2005-11-24
pubmed:abstractText
In the present study, we sought to identify mechanisms underlying increased oxidative stress in vascular tissue in an experimental animal model of chronic congestive heart failure (CHF).
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
http://linkedlifedata.com/resource/pubmed/chemical/Angiotensin-Converting Enzyme..., http://linkedlifedata.com/resource/pubmed/chemical/Captopril, http://linkedlifedata.com/resource/pubmed/chemical/Cell Adhesion Molecules, http://linkedlifedata.com/resource/pubmed/chemical/Guanylate Cyclase, http://linkedlifedata.com/resource/pubmed/chemical/Microfilament Proteins, http://linkedlifedata.com/resource/pubmed/chemical/NADPH Oxidase, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide, http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type III, http://linkedlifedata.com/resource/pubmed/chemical/Phosphoproteins, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Cytoplasmic and Nuclear, http://linkedlifedata.com/resource/pubmed/chemical/Superoxides, http://linkedlifedata.com/resource/pubmed/chemical/neutrophil cytosol factor 67K, http://linkedlifedata.com/resource/pubmed/chemical/soluble guanylyl cyclase, http://linkedlifedata.com/resource/pubmed/chemical/vasodilator-stimulated...
pubmed:status
MEDLINE
pubmed:month
Dec
pubmed:issn
1524-4636
pubmed:author
pubmed:issnType
Electronic
pubmed:volume
25
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2554-9
pubmed:dateRevised
2007-11-15
pubmed:meshHeading
pubmed-meshheading:16224052-Angiotensin-Converting Enzyme Inhibitors, pubmed-meshheading:16224052-Animals, pubmed-meshheading:16224052-Body Weight, pubmed-meshheading:16224052-Captopril, pubmed-meshheading:16224052-Cardiomyopathy, Dilated, pubmed-meshheading:16224052-Cell Adhesion Molecules, pubmed-meshheading:16224052-Cricetinae, pubmed-meshheading:16224052-Disease Models, Animal, pubmed-meshheading:16224052-Female, pubmed-meshheading:16224052-Guanylate Cyclase, pubmed-meshheading:16224052-Heart Failure, pubmed-meshheading:16224052-Male, pubmed-meshheading:16224052-Mesocricetus, pubmed-meshheading:16224052-Microfilament Proteins, pubmed-meshheading:16224052-Myocardium, pubmed-meshheading:16224052-NADPH Oxidase, pubmed-meshheading:16224052-Nitric Oxide, pubmed-meshheading:16224052-Nitric Oxide Synthase Type III, pubmed-meshheading:16224052-Organ Size, pubmed-meshheading:16224052-Oxidative Stress, pubmed-meshheading:16224052-Phosphoproteins, pubmed-meshheading:16224052-Receptors, Cytoplasmic and Nuclear, pubmed-meshheading:16224052-Renin-Angiotensin System, pubmed-meshheading:16224052-Superoxides
pubmed:year
2005
pubmed:articleTitle
Mechanisms of increased vascular superoxide production in an experimental model of idiopathic dilated cardiomyopathy.
pubmed:affiliation
Medizinische Klinik II, Johannes Gutenberg Universität, Mainz, Germany.
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't