Linked Life Data

16180344

Source:http://linkedlifedata.com/resource/pubmed/id/16180344

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
2
pubmed:dateCreated
2005-9-26
pubmed:abstractText
Pathologies of senescence, in particular those of neurosensory organs represent an important health problem. The improvement of the life expectation entails the fast increase of the frequency of the presbyacusis in the population. The biological and molecular causes of this degenerative pathology of the inner ear are linked to the disappearance of the sensory cells (inner and outer hair cells) and are associated to nervous damages of the spiral ganglion in the cochlea. We were interested in mechanisms causing the cochlear degeneration in a model of mouse CD 1 presenting prematurely auditive losses.
pubmed:language
fre
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:issn
0035-1334
pubmed:author
pubmed:issnType
Print
pubmed:volume
126
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
67-74
pubmed:dateRevised
2006-11-15
pubmed:meshHeading
pubmed:year
2005
pubmed:articleTitle
[Implication of mitochondrial apoptosis in neural degeneration of cochlea in a murine model for presbycusis].
pubmed:affiliation
Laboratoire d'Otologie Neuro-Otologie et Microendoscopie Université de la Méditerranée Aix-Marseille II, IFR Jean Roche, Faculté de Médecine Nord, Boulevard Pierre Dramard, F-13916 Marseille cedex 20, France. riva-lavieille.c@jean-roche.univ-mrs.fr
pubmed:publicationType
Journal Article, English Abstract