16126709

Source:http://linkedlifedata.com/resource/pubmed/id/16126709

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0014406, umls-concept:C0162638, umls-concept:C1562025
pubmed:issue	3
pubmed:dateCreated	2005-8-29
pubmed:abstractText	Programmed cell death (PCD) research in higher plants has blossomed in the past ten years. Many evidences suggested that reactive oxygen species,ethylene, Ca2+, salicylic acid, nitric oxide etc. are important signal molecules during environmental stress-induced PCD in plants. Like apoptosis in animals, there also exists a Caspase-dependent PCD signal transduction pathway, in which mitochondrion plays a role of central depot.
pubmed:language	chi
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/9436478
pubmed:status	PubMed-not-MEDLINE
pubmed:month	May
pubmed:issn	0253-9772
pubmed:author	pubmed-author:ChenHongH, pubmed-author:PanJian-WeiJW, pubmed-author:RIBIAA, pubmed-author:ZhuMu-YuanMY
pubmed:issnType	Print
pubmed:volume	24
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	385-8
pubmed:dateRevised	2006-11-15
pubmed:year	2002
pubmed:articleTitle	[Environmental stress-induced programmed cell death in higher plants].
pubmed:affiliation	College of Life Sciences, Zhejiang University, Hangzhou 310012, China.
pubmed:publicationType	Journal Article, English Abstract