| pubmed-article:16103004 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C0026339 | lld:lifeskim |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C0025914 | lld:lifeskim |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C0026809 | lld:lifeskim |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C0011860 | lld:lifeskim |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C2603343 | lld:lifeskim |
| pubmed-article:16103004 | lifeskim:mentions | umls-concept:C1706214 | lld:lifeskim |
| pubmed-article:16103004 | pubmed:issue | 5-6 | lld:pubmed |
| pubmed-article:16103004 | pubmed:dateCreated | 2006-3-13 | lld:pubmed |
| pubmed-article:16103004 | pubmed:abstractText | Obesity and Type 2 diabetes have become epidemics in the Western world. Understanding the pathophysiology of the disease should help in prevention and treatment of these disorders. A common theme is the presence of insulin resistance that eventually results in Type 2 diabetes. To understand the underlying mechanisms in the progression of the disease states, investigators have created mouse models by transgenic overexpression of a candidate gene or produced gene-deletion mouse models. This review will summarize many of the more appropriate models that study insulin resistance and Type 2 diabetes. | lld:pubmed |
| pubmed-article:16103004 | pubmed:language | eng | lld:pubmed |
| pubmed-article:16103004 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16103004 | pubmed:citationSubset | IM | lld:pubmed |
| pubmed-article:16103004 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16103004 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16103004 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
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| pubmed-article:16103004 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:16103004 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:16103004 | pubmed:issn | 1357-2725 | lld:pubmed |
| pubmed-article:16103004 | pubmed:author | pubmed-author:LeRoithDerekD | lld:pubmed |
| pubmed-article:16103004 | pubmed:author | pubmed-author:GavrilovaOksa... | lld:pubmed |
| pubmed-article:16103004 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:16103004 | pubmed:volume | 38 | lld:pubmed |
| pubmed-article:16103004 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:16103004 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:16103004 | pubmed:pagination | 904-12 | lld:pubmed |
| pubmed-article:16103004 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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| pubmed-article:16103004 | pubmed:meshHeading | pubmed-meshheading:16103004... | lld:pubmed |
| pubmed-article:16103004 | pubmed:year | 2006 | lld:pubmed |
| pubmed-article:16103004 | pubmed:articleTitle | Mouse models created to study the pathophysiology of Type 2 diabetes. | lld:pubmed |
| pubmed-article:16103004 | pubmed:affiliation | Diabetes Branch, National Institutes of Health, Room 8D12, Building 10, Bethesda, MD 20892-1758, USA. derek@helix.nih.gov | lld:pubmed |
| pubmed-article:16103004 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:16103004 | pubmed:publicationType | Review | lld:pubmed |
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