pubmed-article:15987275 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:15987275 | lifeskim:mentions | umls-concept:C0178499 | lld:lifeskim |
pubmed-article:15987275 | lifeskim:mentions | umls-concept:C0205210 | lld:lifeskim |
pubmed-article:15987275 | lifeskim:mentions | umls-concept:C2349101 | lld:lifeskim |
pubmed-article:15987275 | lifeskim:mentions | umls-concept:C0441712 | lld:lifeskim |
pubmed-article:15987275 | lifeskim:mentions | umls-concept:C1527178 | lld:lifeskim |
pubmed-article:15987275 | lifeskim:mentions | umls-concept:C0205923 | lld:lifeskim |
pubmed-article:15987275 | pubmed:dateCreated | 2005-6-30 | lld:pubmed |
pubmed-article:15987275 | pubmed:abstractText | Gamma-aminobutyric acid (GABA)-A/benzodiazepine receptors (BZRs) play an important inhibitory role in epileptogenesis. [123I]Iomazenil (123I-IMZ) is a specific ligand for central-type (or neuronal-type) BNRs and is available for single-photon emission computed tomography (SPECT) in brain disorders. We demonstrated alterations of central-type BZRs in human focal epilepsies and their experimental models. | lld:pubmed |
pubmed-article:15987275 | pubmed:language | eng | lld:pubmed |
pubmed-article:15987275 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15987275 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:15987275 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15987275 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15987275 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15987275 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15987275 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15987275 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15987275 | pubmed:issn | 0013-9580 | lld:pubmed |
pubmed-article:15987275 | pubmed:author | pubmed-author:MorimotoKiyos... | lld:pubmed |
pubmed-article:15987275 | pubmed:author | pubmed-author:MatsudaKazumi... | lld:pubmed |
pubmed-article:15987275 | pubmed:author | pubmed-author:TamagamiHiros... | lld:pubmed |
pubmed-article:15987275 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15987275 | pubmed:volume | 46 Suppl 5 | lld:pubmed |
pubmed-article:15987275 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15987275 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15987275 | pubmed:pagination | 184-8 | lld:pubmed |
pubmed-article:15987275 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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pubmed-article:15987275 | pubmed:year | 2005 | lld:pubmed |
pubmed-article:15987275 | pubmed:articleTitle | Central-type benzodiazepine receptors and epileptogenesis: basic mechanisms and clinical validity. | lld:pubmed |
pubmed-article:15987275 | pubmed:affiliation | Department of Neuropsychiatry, Faculty of Medicine, Kagawa Medical University, Kagawa, Japan. neuropsy@kms.ac.jp | lld:pubmed |
pubmed-article:15987275 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15987275 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |