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pubmed-article:15955029pubmed:abstractText1. The pressor action of noradrenaline and its blockade by selective alpha(1)-adrenoceptor antagonists in the pithed mouse were evaluated. 2. Chloroethylclonidine (alpha(1B/D)-adrenoceptor alkylating agent) or BMY 7378 (alpha(1D)-adrenoceptor antagonist), both at 1 mg kg(-1), did not block the increase in blood pressure induced by noradrenaline. 3. 5-Methylurapidil (alpha(1A)-adrenoceptor antagonist), at 0.1 mg kg(-1), displaced the dose-response curve approximately six-fold to the right. 4. The results support the idea that the pithed mouse vasculature express alpha(1A)-adrenoceptors and suggest that it is a good model to study the roles of alpha(1)-adrenoceptors in gene knockout or overexpression.lld:pubmed
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pubmed-article:15955029pubmed:dateRevised2010-11-18lld:pubmed
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pubmed-article:15955029pubmed:articleTitlePostjunctional alpha1-adrenoceptors in the vasculature of the pithed mouse are of the alpha1A-subtype.lld:pubmed
pubmed-article:15955029pubmed:affiliationDepartamento de Farmacobiología, Centro de Investigación y de Estudios Avanzados, Sede Sur, México D.F.lld:pubmed
pubmed-article:15955029pubmed:publicationTypeJournal Articlelld:pubmed
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