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pubmed-article:15949725pubmed:abstractTextThe majority of cyclosporine-treated transplant recipients develop hypertension. Endothelin-1 (ET-1) has been suggested to mediate cyclosporine-induced vasoconstriction when binding to ET-A receptors. We hypothesized that cyclosporine-treated lung transplant recipients have an increased basal vascular resistance and an augmented response to ET-A receptor blockade.lld:pubmed
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pubmed-article:15949725pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:15949725pubmed:articleTitleBlunted vascular response to endothelin-a receptor blockade in cyclosporine-treated lung transplant recipients.lld:pubmed
pubmed-article:15949725pubmed:affiliationDepartment of Cardiothoracic Surgery, Sahlgrenska University Hospital, Gothenburg, Sweden.lld:pubmed
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