| pubmed-article:15914639 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C0026336 | lld:lifeskim |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C0035309 | lld:lifeskim |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C0035320 | lld:lifeskim |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C1749467 | lld:lifeskim |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C0334094 | lld:lifeskim |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C1414431 | lld:lifeskim |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C0392756 | lld:lifeskim |
| pubmed-article:15914639 | lifeskim:mentions | umls-concept:C0376315 | lld:lifeskim |
| pubmed-article:15914639 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:15914639 | pubmed:dateCreated | 2005-5-25 | lld:pubmed |
| pubmed-article:15914639 | pubmed:abstractText | Ephrin ligands and their Eph receptors are key regulators of endothelial cell (EC) proliferation, migration, adhesion, and repulsion during mammalian vascular development. The hypothesis was that these molecules also play a role in pathologic neovascularization (NV) in the mouse model of oxygen-induced retinopathy. | lld:pubmed |
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| pubmed-article:15914639 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15914639 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15914639 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15914639 | pubmed:month | Jun | lld:pubmed |
| pubmed-article:15914639 | pubmed:issn | 0146-0404 | lld:pubmed |
| pubmed-article:15914639 | pubmed:author | pubmed-author:RosenbaumJame... | lld:pubmed |
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| pubmed-article:15914639 | pubmed:author | pubmed-author:ZamoraDavid... | lld:pubmed |
| pubmed-article:15914639 | pubmed:author | pubmed-author:DaviesMichael... | lld:pubmed |
| pubmed-article:15914639 | pubmed:author | pubmed-author:PowersMichael... | lld:pubmed |
| pubmed-article:15914639 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15914639 | pubmed:volume | 46 | lld:pubmed |
| pubmed-article:15914639 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15914639 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15914639 | pubmed:pagination | 2175-82 | lld:pubmed |
| pubmed-article:15914639 | pubmed:dateRevised | 2009-11-19 | lld:pubmed |
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| pubmed-article:15914639 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15914639 | pubmed:articleTitle | Soluble forms of EphrinB2 and EphB4 reduce retinal neovascularization in a model of proliferative retinopathy. | lld:pubmed |
| pubmed-article:15914639 | pubmed:affiliation | Department of Ophthalmology, Casey Eye Institute, Oregon Health & Science University, Portland, 97239, USA. | lld:pubmed |
| pubmed-article:15914639 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15914639 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:15914639 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
| pubmed-article:15914639 | pubmed:publicationType | Research Support, N.I.H., Extramural | lld:pubmed |
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