Linked Life Data

15914639

Source:http://linkedlifedata.com/resource/pubmed/id/15914639

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
6
pubmed:dateCreated
2005-5-25
pubmed:abstractText
Ephrin ligands and their Eph receptors are key regulators of endothelial cell (EC) proliferation, migration, adhesion, and repulsion during mammalian vascular development. The hypothesis was that these molecules also play a role in pathologic neovascularization (NV) in the mouse model of oxygen-induced retinopathy.
pubmed:grant
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jun
pubmed:issn
0146-0404
pubmed:author
pubmed:issnType
Print
pubmed:volume
46
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
2175-82
pubmed:dateRevised
2009-11-19
pubmed:meshHeading
pubmed-meshheading:15914639-Animals, pubmed-meshheading:15914639-Animals, Newborn, pubmed-meshheading:15914639-Disease Models, Animal, pubmed-meshheading:15914639-Ephrin-B2, pubmed-meshheading:15914639-Gene Expression, pubmed-meshheading:15914639-Humans, pubmed-meshheading:15914639-Hyperoxia, pubmed-meshheading:15914639-Immunohistochemistry, pubmed-meshheading:15914639-Infant, Newborn, pubmed-meshheading:15914639-Injections, pubmed-meshheading:15914639-Mice, pubmed-meshheading:15914639-Mice, Inbred C57BL, pubmed-meshheading:15914639-Oxygen, pubmed-meshheading:15914639-RNA, Messenger, pubmed-meshheading:15914639-Receptor, EphB4, pubmed-meshheading:15914639-Retinal Neovascularization, pubmed-meshheading:15914639-Retinal Vessels, pubmed-meshheading:15914639-Retinopathy of Prematurity, pubmed-meshheading:15914639-Reverse Transcriptase Polymerase Chain Reaction, pubmed-meshheading:15914639-Solubility
pubmed:year
2005
pubmed:articleTitle
Soluble forms of EphrinB2 and EphB4 reduce retinal neovascularization in a model of proliferative retinopathy.
pubmed:affiliation
Department of Ophthalmology, Casey Eye Institute, Oregon Health & Science University, Portland, 97239, USA.
pubmed:publicationType
Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't, Research Support, N.I.H., Extramural