|
rdf:type |
|
|
lifeskim:mentions |
umls-concept:C0012155,
umls-concept:C0020459,
umls-concept:C0025914,
umls-concept:C0026809,
umls-concept:C0032105,
umls-concept:C0050156,
umls-concept:C0271706,
umls-concept:C0299583,
umls-concept:C0332281,
umls-concept:C0389071,
umls-concept:C0441889,
umls-concept:C0547047,
umls-concept:C1444748
|
|
pubmed:issue |
6
|
|
pubmed:dateCreated |
2005-6-16
|
|
pubmed:abstractText |
Dietary supplementation with conjugated linoleic acids (CLA) has a fat-reducing effect in various species, but induces severe hyperinsulinaemia and hepatic steatosis in the mouse. This study aimed to determine the causes of the deleterious effects of CLA on insulin homeostasis.
|
|
pubmed:commentsCorrections |
|
|
pubmed:language |
eng
|
|
pubmed:journal |
|
|
pubmed:citationSubset |
IM
|
|
pubmed:chemical |
|
|
pubmed:status |
MEDLINE
|
|
pubmed:month |
Jun
|
|
pubmed:issn |
0012-186X
|
|
pubmed:author |
|
|
pubmed:issnType |
Print
|
|
pubmed:volume |
48
|
|
pubmed:owner |
NLM
|
|
pubmed:authorsComplete |
Y
|
|
pubmed:pagination |
1059-65
|
|
pubmed:dateRevised |
2011-11-17
|
|
pubmed:meshHeading |
pubmed-meshheading:15868135-Adiponectin,
pubmed-meshheading:15868135-Adipose Tissue,
pubmed-meshheading:15868135-Animals,
pubmed-meshheading:15868135-Body Weight,
pubmed-meshheading:15868135-Disease Models, Animal,
pubmed-meshheading:15868135-Female,
pubmed-meshheading:15868135-Hyperinsulinism,
pubmed-meshheading:15868135-Hyperplasia,
pubmed-meshheading:15868135-Insulin,
pubmed-meshheading:15868135-Intercellular Signaling Peptides and Proteins,
pubmed-meshheading:15868135-Islets of Langerhans,
pubmed-meshheading:15868135-Leptin,
pubmed-meshheading:15868135-Linoleic Acids, Conjugated,
pubmed-meshheading:15868135-Liver,
pubmed-meshheading:15868135-Mice,
pubmed-meshheading:15868135-Mice, Inbred C57BL,
pubmed-meshheading:15868135-Organ Size,
pubmed-meshheading:15868135-Triglycerides
|
|
pubmed:year |
2005
|
|
pubmed:articleTitle |
Hyperinsulinaemia triggered by dietary conjugated linoleic acid is associated with a decrease in leptin and adiponectin plasma levels and pancreatic beta cell hyperplasia in the mouse.
|
|
pubmed:affiliation |
Department of Nutritional Physiology, ENSBANA, UMR 5170 CNRS-CESG/1214INRA/University of Burgundy, Dijon, France.
|
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|
