Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
1
pubmed:dateCreated
2005-2-7
pubmed:abstractText
The invasive pathogen Salmonella enterica has evolved sophisticated mechanisms to subvert the cytoskeletal machinery of its host. Following contact with the host cell, it delivers a distinct arsenal of effector proteins directly into the cytoplasm. These bacterial effectors coordinate transient actin rearrangements and alter vesicle trafficking to trigger invasion, without causing overt cellular damage. Recent studies have shed new light on the signaling mechanisms underlying this remarkable host-pathogen interface, in particular, highlighting the unique multi-functional role and temporal regulation of key bacterial effectors.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Feb
pubmed:issn
1369-5274
pubmed:author
pubmed:issnType
Print
pubmed:volume
8
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
10-5
pubmed:dateRevised
2005-11-16
pubmed:meshHeading
pubmed:year
2005
pubmed:articleTitle
Manipulation of the host actin cytoskeleton by Salmonella--all in the name of entry.
pubmed:affiliation
Section of Microbial Pathogenesis, Yale University School of Medicine, New Haven, CT 06536, USA.
pubmed:publicationType
Journal Article, Review