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| pubmed-article:15692095 | lifeskim:mentions | umls-concept:C0014257 | lld:lifeskim |
| pubmed-article:15692095 | lifeskim:mentions | umls-concept:C0004153 | lld:lifeskim |
| pubmed-article:15692095 | lifeskim:mentions | umls-concept:C0242606 | lld:lifeskim |
| pubmed-article:15692095 | lifeskim:mentions | umls-concept:C0277785 | lld:lifeskim |
| pubmed-article:15692095 | lifeskim:mentions | umls-concept:C0442805 | lld:lifeskim |
| pubmed-article:15692095 | lifeskim:mentions | umls-concept:C0205485 | lld:lifeskim |
| pubmed-article:15692095 | pubmed:issue | 4 | lld:pubmed |
| pubmed-article:15692095 | pubmed:dateCreated | 2005-3-25 | lld:pubmed |
| pubmed-article:15692095 | pubmed:abstractText | Sedentary lifestyle is associated with increased cardiovascular events. The underlying molecular mechanisms are incompletely understood. Reactive oxygen species (ROS) contribute to endothelial dysfunction and atherosclerosis. An important source of vascular ROS is the NADPH oxidase. | lld:pubmed |
| pubmed-article:15692095 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15692095 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15692095 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15692095 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15692095 | pubmed:month | Apr | lld:pubmed |
| pubmed-article:15692095 | pubmed:issn | 1524-4636 | lld:pubmed |
| pubmed-article:15692095 | pubmed:author | pubmed-author:BöhmMichaelM | lld:pubmed |
| pubmed-article:15692095 | pubmed:author | pubmed-author:NickenigGeorg... | lld:pubmed |
| pubmed-article:15692095 | pubmed:author | pubmed-author:CzechThomasT | lld:pubmed |
| pubmed-article:15692095 | pubmed:author | pubmed-author:WassmannSvenS | lld:pubmed |
| pubmed-article:15692095 | pubmed:author | pubmed-author:LaufsUlrichU | lld:pubmed |
| pubmed-article:15692095 | pubmed:author | pubmed-author:MünzelThomasT | lld:pubmed |
| pubmed-article:15692095 | pubmed:author | pubmed-author:EisenhauerMar... | lld:pubmed |
| pubmed-article:15692095 | pubmed:issnType | Electronic | lld:pubmed |
| pubmed-article:15692095 | pubmed:volume | 25 | lld:pubmed |
| pubmed-article:15692095 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15692095 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15692095 | pubmed:pagination | 809-14 | lld:pubmed |
| pubmed-article:15692095 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
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| pubmed-article:15692095 | pubmed:year | 2005 | lld:pubmed |
| pubmed-article:15692095 | pubmed:articleTitle | Physical inactivity increases oxidative stress, endothelial dysfunction, and atherosclerosis. | lld:pubmed |
| pubmed-article:15692095 | pubmed:affiliation | Klinik für Innere Medizin III, niversitätsklinikum des Saarlandes, Homburg/Saar, Germany. ulrich@laufs.com | lld:pubmed |
| pubmed-article:15692095 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15692095 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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