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rdf:type |
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lifeskim:mentions |
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pubmed:issue |
4
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pubmed:dateCreated |
2005-3-25
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pubmed:abstractText |
Sedentary lifestyle is associated with increased cardiovascular events. The underlying molecular mechanisms are incompletely understood. Reactive oxygen species (ROS) contribute to endothelial dysfunction and atherosclerosis. An important source of vascular ROS is the NADPH oxidase.
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pubmed:language |
eng
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pubmed:journal |
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pubmed:citationSubset |
IM
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pubmed:chemical |
http://linkedlifedata.com/resource/pubmed/chemical/Apolipoproteins E,
http://linkedlifedata.com/resource/pubmed/chemical/NADH, NADPH Oxidoreductases,
http://linkedlifedata.com/resource/pubmed/chemical/NADPH Oxidase,
http://linkedlifedata.com/resource/pubmed/chemical/NADPH oxidase 1,
http://linkedlifedata.com/resource/pubmed/chemical/Neuropeptides,
http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase,
http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type II,
http://linkedlifedata.com/resource/pubmed/chemical/Nitric Oxide Synthase Type III,
http://linkedlifedata.com/resource/pubmed/chemical/Nos3 protein, mouse,
http://linkedlifedata.com/resource/pubmed/chemical/Phosphoproteins,
http://linkedlifedata.com/resource/pubmed/chemical/RNA, Messenger,
http://linkedlifedata.com/resource/pubmed/chemical/Rac1 protein, mouse,
http://linkedlifedata.com/resource/pubmed/chemical/Reactive Oxygen Species,
http://linkedlifedata.com/resource/pubmed/chemical/neutrophil cytosol factor 67K,
http://linkedlifedata.com/resource/pubmed/chemical/neutrophil cytosolic factor 1,
http://linkedlifedata.com/resource/pubmed/chemical/rac GTP-Binding Proteins
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pubmed:status |
MEDLINE
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pubmed:month |
Apr
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pubmed:issn |
1524-4636
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pubmed:author |
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pubmed:issnType |
Electronic
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pubmed:volume |
25
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
809-14
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pubmed:dateRevised |
2006-11-15
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pubmed:meshHeading |
pubmed-meshheading:15692095-Animals,
pubmed-meshheading:15692095-Apolipoproteins E,
pubmed-meshheading:15692095-Arteriosclerosis,
pubmed-meshheading:15692095-Endothelium, Vascular,
pubmed-meshheading:15692095-Life Style,
pubmed-meshheading:15692095-Mice,
pubmed-meshheading:15692095-Mice, Inbred C57BL,
pubmed-meshheading:15692095-Mice, Mutant Strains,
pubmed-meshheading:15692095-NADH, NADPH Oxidoreductases,
pubmed-meshheading:15692095-NADPH Oxidase,
pubmed-meshheading:15692095-Neuropeptides,
pubmed-meshheading:15692095-Nitric Oxide Synthase,
pubmed-meshheading:15692095-Nitric Oxide Synthase Type II,
pubmed-meshheading:15692095-Nitric Oxide Synthase Type III,
pubmed-meshheading:15692095-Oxidative Stress,
pubmed-meshheading:15692095-Phosphoproteins,
pubmed-meshheading:15692095-Physical Conditioning, Animal,
pubmed-meshheading:15692095-RNA, Messenger,
pubmed-meshheading:15692095-Reactive Oxygen Species,
pubmed-meshheading:15692095-Vasodilation,
pubmed-meshheading:15692095-rac GTP-Binding Proteins
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pubmed:year |
2005
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pubmed:articleTitle |
Physical inactivity increases oxidative stress, endothelial dysfunction, and atherosclerosis.
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pubmed:affiliation |
Klinik für Innere Medizin III, niversitätsklinikum des Saarlandes, Homburg/Saar, Germany. ulrich@laufs.com
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pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
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