| pubmed-article:1563790 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1563790 | lifeskim:mentions | umls-concept:C0040558 | lld:lifeskim |
| pubmed-article:1563790 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:1563790 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
| pubmed-article:1563790 | lifeskim:mentions | umls-concept:C0205227 | lld:lifeskim |
| pubmed-article:1563790 | pubmed:issue | 5 | lld:pubmed |
| pubmed-article:1563790 | pubmed:dateCreated | 1992-5-19 | lld:pubmed |
| pubmed-article:1563790 | pubmed:abstractText | The involvement of tumor necrosis factor (TNF) in resistance to Toxoplasma gondii infection was examined by means of experiments in which mice were treated with anti-TNF antibodies prior to infection with ME49, a low-virulence Toxoplasma strain. In (BALB/cBy x C57BL/6J)F1 (CB6F1) mice, which are highly resistant to intraperitoneal (i.p.) infection with T. gondii ME49, 10(4) neutralizing units of anti-TNF caused a significant increase in trophozoite numbers in the peritoneal cavities of infected mice and transient signs of illness but no deaths. i.p. infection of anti-TNF-treated C57BL/6J (B6) mice, which are more susceptible to T. gondii and develop a chronic progressive toxoplasmosis, resulted in death for some of the mice. If the mice were infected perorally, however, and treated with anti-TNF, mortality was extensive in B6 mice but not in CB6F1 mice. Although it was not detected in their sera, TNF was found in the peritoneal fluids of i.p.-infected CB6F1 and B6 mice. Endogenously produced TNF thus appears to be an important mediator of resistance to T. gondii. | lld:pubmed |
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| pubmed-article:1563790 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1563790 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1563790 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1563790 | pubmed:month | May | lld:pubmed |
| pubmed-article:1563790 | pubmed:issn | 0019-9567 | lld:pubmed |
| pubmed-article:1563790 | pubmed:author | pubmed-author:JohnsonL LLL | lld:pubmed |
| pubmed-article:1563790 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1563790 | pubmed:volume | 60 | lld:pubmed |
| pubmed-article:1563790 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1563790 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1563790 | pubmed:pagination | 1979-83 | lld:pubmed |
| pubmed-article:1563790 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
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| pubmed-article:1563790 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1563790 | pubmed:articleTitle | A protective role for endogenous tumor necrosis factor in Toxoplasma gondii infection. | lld:pubmed |
| pubmed-article:1563790 | pubmed:affiliation | Trudeau Institute, Inc., Saranac Lake, New York 12983. | lld:pubmed |
| pubmed-article:1563790 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1563790 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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