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| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C0238339 | lld:lifeskim |
| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C0017337 | lld:lifeskim |
| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C0017837 | lld:lifeskim |
| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C0919534 | lld:lifeskim |
| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C0796344 | lld:lifeskim |
| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C1333715 | lld:lifeskim |
| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C1415336 | lld:lifeskim |
| pubmed-article:15338373 | lifeskim:mentions | umls-concept:C1417145 | lld:lifeskim |
| pubmed-article:15338373 | pubmed:issue | 8 | lld:pubmed |
| pubmed-article:15338373 | pubmed:dateCreated | 2004-8-31 | lld:pubmed |
| pubmed-article:15338373 | pubmed:abstractText | Specific mutations in the cationic trypsinogen gene ( PRSS1) are disease-causing in patients with hereditary pancreatitis, but the genetic background still remains mysterious in about 40% of patients with the disease. It has been suggested that oxidative stress contributes to pancreatic damage. The glutathione s-transferases (GSTs) represent major detoxification enzymes that protect cells from oxidative stress. | lld:pubmed |
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| pubmed-article:15338373 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:15338373 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15338373 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15338373 | pubmed:month | Aug | lld:pubmed |
| pubmed-article:15338373 | pubmed:issn | 0944-1174 | lld:pubmed |
| pubmed-article:15338373 | pubmed:author | pubmed-author:BarmadaM... | lld:pubmed |
| pubmed-article:15338373 | pubmed:author | pubmed-author:WhitcombDavid... | lld:pubmed |
| pubmed-article:15338373 | pubmed:author | pubmed-author:SchneiderAlex... | lld:pubmed |
| pubmed-article:15338373 | pubmed:author | pubmed-author:TögelSandraS | lld:pubmed |
| pubmed-article:15338373 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:15338373 | pubmed:volume | 39 | lld:pubmed |
| pubmed-article:15338373 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15338373 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15338373 | pubmed:pagination | 783-7 | lld:pubmed |
| pubmed-article:15338373 | pubmed:dateRevised | 2007-11-14 | lld:pubmed |
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| pubmed-article:15338373 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15338373 | pubmed:articleTitle | Genetic analysis of the glutathione s-transferase genes MGST1, GSTM3, GSTT1, and GSTM1 in patients with hereditary pancreatitis. | lld:pubmed |
| pubmed-article:15338373 | pubmed:affiliation | Department of Medicine, Division of Gastroenterology, University of Pittsburgh, 571 Scaife Hall, 3550 Terrace Street, Pittsburgh, PA 15213, USA. | lld:pubmed |
| pubmed-article:15338373 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:15338373 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
| pubmed-article:15338373 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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