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| pubmed-article:15177461 | lifeskim:mentions | umls-concept:C0007226 | lld:lifeskim |
| pubmed-article:15177461 | lifeskim:mentions | umls-concept:C0243192 | lld:lifeskim |
| pubmed-article:15177461 | lifeskim:mentions | umls-concept:C0205178 | lld:lifeskim |
| pubmed-article:15177461 | lifeskim:mentions | umls-concept:C1414255 | lld:lifeskim |
| pubmed-article:15177461 | lifeskim:mentions | umls-concept:C2349975 | lld:lifeskim |
| pubmed-article:15177461 | lifeskim:mentions | umls-concept:C0390526 | lld:lifeskim |
| pubmed-article:15177461 | lifeskim:mentions | umls-concept:C1565169 | lld:lifeskim |
| pubmed-article:15177461 | pubmed:issue | 13 | lld:pubmed |
| pubmed-article:15177461 | pubmed:dateCreated | 2004-6-4 | lld:pubmed |
| pubmed-article:15177461 | pubmed:abstractText | Structurally modified 3-(N-benzylamino)propylphosphonic acid S1P receptor agonists that maintain affinity for S1P1, and have decreased affinity for S1P3 are efficacious, but exhibit decreased acute cardiovascular toxicity in rodents than do nonselective agonists. | lld:pubmed |
| pubmed-article:15177461 | pubmed:language | eng | lld:pubmed |
| pubmed-article:15177461 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:15177461 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:15177461 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:15177461 | pubmed:month | Jul | lld:pubmed |
| pubmed-article:15177461 | pubmed:issn | 0960-894X | lld:pubmed |
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| pubmed-article:15177461 | pubmed:issnType | Print | lld:pubmed |
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| pubmed-article:15177461 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:15177461 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:15177461 | pubmed:pagination | 3501-5 | lld:pubmed |
| pubmed-article:15177461 | pubmed:dateRevised | 2005-11-17 | lld:pubmed |
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| pubmed-article:15177461 | pubmed:year | 2004 | lld:pubmed |
| pubmed-article:15177461 | pubmed:articleTitle | Selecting against S1P3 enhances the acute cardiovascular tolerability of 3-(N-benzyl)aminopropylphosphonic acid S1P receptor agonists. | lld:pubmed |
| pubmed-article:15177461 | pubmed:affiliation | Department of Medicinal Chemistry, Merck Research Laboratories, Rahway, NJ 07065, USA. jeffrey_hale@merck.com | lld:pubmed |
| pubmed-article:15177461 | pubmed:publicationType | Journal Article | lld:pubmed |
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