15118592

Source:http://linkedlifedata.com/resource/pubmed/id/15118592

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Predicate	Object
rdf:type	pubmed:Citation
lifeskim:mentions	umls-concept:C0021755, umls-concept:C0220783, umls-concept:C0597357, umls-concept:C0806987
pubmed:issue	5
pubmed:dateCreated	2004-4-30
pubmed:abstractText	The anaphylatoxins, C3a and C5a, that are generated during trauma, major surgery, or infection are potent proinflammatory mediators that increase interleukin (IL-1) cytokine synthesis. We investigated the effects of IL-1 on anaphylatoxin receptor expression in monocytes.
pubmed:grant	http://linkedlifedata.com/resource/pubmed/grant/GM-21700, http://linkedlifedata.com/resource/pubmed/grant/GM-50766
pubmed:language	eng
pubmed:journal	http://linkedlifedata.com/resource/pubmed/journal/0417347
pubmed:citationSubset	AIM
pubmed:chemical	http://linkedlifedata.com/resource/pubmed/chemical/Anaphylatoxins, http://linkedlifedata.com/resource/pubmed/chemical/Calcium, http://linkedlifedata.com/resource/pubmed/chemical/Complement C3a, http://linkedlifedata.com/resource/pubmed/chemical/Complement C5a, http://linkedlifedata.com/resource/pubmed/chemical/IL1RN protein, human, http://linkedlifedata.com/resource/pubmed/chemical/Interleukin 1 Receptor Antagonist..., http://linkedlifedata.com/resource/pubmed/chemical/Interleukin-1, http://linkedlifedata.com/resource/pubmed/chemical/Macrophage-1 Antigen, http://linkedlifedata.com/resource/pubmed/chemical/Pertussis Toxin, http://linkedlifedata.com/resource/pubmed/chemical/Receptor, Anaphylatoxin C5a, http://linkedlifedata.com/resource/pubmed/chemical/Receptors, Cell Surface, http://linkedlifedata.com/resource/pubmed/chemical/Recombinant Proteins, http://linkedlifedata.com/resource/pubmed/chemical/Sialoglycoproteins
pubmed:status	MEDLINE
pubmed:month	May
pubmed:issn	0039-6060
pubmed:author	pubmed-author:BeinbornMartinM, pubmed-author:BurkeJohn FJF, pubmed-author:ClarkBurton DBD, pubmed-author:GelfandJeffrey AJA, pubmed-author:ShimizuSoichiS, pubmed-author:TakabayashiTsukasaT
pubmed:issnType	Print
pubmed:volume	135
pubmed:owner	NLM
pubmed:authorsComplete	Y
pubmed:pagination	544-54
pubmed:dateRevised	2007-11-14
pubmed:meshHeading	pubmed-meshheading:15118592-Anaphylatoxins, pubmed-meshheading:15118592-Binding Sites, pubmed-meshheading:15118592-Calcium, pubmed-meshheading:15118592-Cell Line, pubmed-meshheading:15118592-Complement C3a, pubmed-meshheading:15118592-Complement C5a, pubmed-meshheading:15118592-Cytosol, pubmed-meshheading:15118592-Dose-Response Relationship, Drug, pubmed-meshheading:15118592-Gene Expression, pubmed-meshheading:15118592-Humans, pubmed-meshheading:15118592-Interleukin 1 Receptor Antagonist Protein, pubmed-meshheading:15118592-Interleukin-1, pubmed-meshheading:15118592-Macrophage-1 Antigen, pubmed-meshheading:15118592-Monocytes, pubmed-meshheading:15118592-Osmolar Concentration, pubmed-meshheading:15118592-Pertussis Toxin, pubmed-meshheading:15118592-Receptor, Anaphylatoxin C5a, pubmed-meshheading:15118592-Receptors, Cell Surface, pubmed-meshheading:15118592-Recombinant Proteins, pubmed-meshheading:15118592-Sialoglycoproteins, pubmed-meshheading:15118592-Up-Regulation
pubmed:year	2004
pubmed:articleTitle	Interleukin-1 upregulates anaphylatoxin receptors on mononuclear cells.
pubmed:affiliation	Department of Surgery, Harvard Medical School and Massachusetts General Hospital, Boston, Massschusetts 02114, USA.
pubmed:publicationType	Journal Article, Research Support, U.S. Gov't, P.H.S., Research Support, Non-U.S. Gov't