pubmed-article:15080267 | rdf:type | pubmed:Citation | lld:pubmed |
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pubmed-article:15080267 | lifeskim:mentions | umls-concept:C0385242 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C1550315 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C1456820 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C1336646 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C0023688 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C0231204 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C0445223 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C0205263 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C1552599 | lld:lifeskim |
pubmed-article:15080267 | lifeskim:mentions | umls-concept:C1704787 | lld:lifeskim |
pubmed-article:15080267 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:15080267 | pubmed:dateCreated | 2004-4-14 | lld:pubmed |
pubmed-article:15080267 | pubmed:abstractText | To investigate the expression of tumour necrosis factor (TNF)-related apoptosis-inducing ligand (TRAIL) receptors in cultured synovial fibroblasts from rheumatoid arthritis (RA) and osteoarthritis (OA) patients, and to examine their susceptibility to TRAIL-induced apoptosis in the presence or absence of metabolic inhibitors. | lld:pubmed |
pubmed-article:15080267 | pubmed:language | eng | lld:pubmed |
pubmed-article:15080267 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:15080267 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:15080267 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:15080267 | pubmed:issn | 0300-9742 | lld:pubmed |
pubmed-article:15080267 | pubmed:author | pubmed-author:LeeJ SJS | lld:pubmed |
pubmed-article:15080267 | pubmed:author | pubmed-author:MessBB | lld:pubmed |
pubmed-article:15080267 | pubmed:author | pubmed-author:RyangD WDW | lld:pubmed |
pubmed-article:15080267 | pubmed:author | pubmed-author:YooD HDH | lld:pubmed |
pubmed-article:15080267 | pubmed:author | pubmed-author:ImJ KJK | lld:pubmed |
pubmed-article:15080267 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:15080267 | pubmed:volume | 32 | lld:pubmed |
pubmed-article:15080267 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:15080267 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:15080267 | pubmed:pagination | 356-63 | lld:pubmed |
pubmed-article:15080267 | pubmed:dateRevised | 2010-11-18 | lld:pubmed |
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pubmed-article:15080267 | pubmed:year | 2003 | lld:pubmed |
pubmed-article:15080267 | pubmed:articleTitle | Actinomycin D renders cultured synovial fibroblasts susceptible to tumour necrosis factor related apoptosis-inducing ligand (TRAIL)-induced apoptosis. | lld:pubmed |
pubmed-article:15080267 | pubmed:affiliation | Division of Rheumatology, The Hospital for Rheumatic Diseases, Hanyang University, Seoul, Korea. | lld:pubmed |
pubmed-article:15080267 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:15080267 | pubmed:publicationType | Comparative Study | lld:pubmed |
pubmed-article:15080267 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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