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pubmed-article:14871429pubmed:abstractTextTarget organs express antigens recognized directly by antigen-specific T cells, and their recognition is crucial to precipitate rejection. Then, the earliest T-cell activation is inhibited by cyclosporine A (CsA), the lowest would be the risk of rejection. Here, we aimed to assess this possibility in a large cohort of de novo kidney transplant recipients participating in an ongoing clinical trial, the Mycophenolate Steroid-Sparing (MY.S.S.) Trial.lld:pubmed
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pubmed-article:14871429pubmed:dateRevised2006-11-15lld:pubmed
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pubmed-article:14871429pubmed:articleTitleIn renal transplantation blood cyclosporine levels soon after surgery act as a major determinant of rejection: insights from the MY.S.S. trial.lld:pubmed
pubmed-article:14871429pubmed:affiliationDepartment of Medicine and Transplantation, Azienda Ospedaliera, Ospedali Riuniti di Bergamo-Mario Negri Institute for Pharmacological Research, Bergamo, Italy.lld:pubmed
pubmed-article:14871429pubmed:publicationTypeJournal Articlelld:pubmed
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pubmed-article:14871429pubmed:publicationTypeResearch Support, Non-U.S. Gov'tlld:pubmed
pubmed-article:14871429pubmed:publicationTypeMulticenter Studylld:pubmed
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