| pubmed-article:1403646 | rdf:type | pubmed:Citation | lld:pubmed |
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| pubmed-article:1403646 | lifeskim:mentions | umls-concept:C0039341 | lld:lifeskim |
| pubmed-article:1403646 | lifeskim:mentions | umls-concept:C0441655 | lld:lifeskim |
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| pubmed-article:1403646 | lifeskim:mentions | umls-concept:C0040649 | lld:lifeskim |
| pubmed-article:1403646 | lifeskim:mentions | umls-concept:C0076923 | lld:lifeskim |
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| pubmed-article:1403646 | lifeskim:mentions | umls-concept:C2755756 | lld:lifeskim |
| pubmed-article:1403646 | pubmed:issue | 11 | lld:pubmed |
| pubmed-article:1403646 | pubmed:dateCreated | 1992-11-10 | lld:pubmed |
| pubmed-article:1403646 | pubmed:abstractText | The HIV Tat protein is able to upregulate the transcription by RNA polymerase III of cotransfected or endogenous cellular Alu-repeated sequences in both HeLa and Jurkat T cells. This effect is mediated by an increase in the activity of transcription factor TFIIIC, which binds to the B box in the RNA polymerase III Alu promoter. This is the first example of an effect of the Tat protein on the transcription of a cellular gene or on the activity of a cellular transcription factor. The significance of this effect for the life cycle of HIV and its interaction with infected cells is discussed. | lld:pubmed |
| pubmed-article:1403646 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:1403646 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1403646 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1403646 | pubmed:issn | 0894-9255 | lld:pubmed |
| pubmed-article:1403646 | pubmed:author | pubmed-author:LatchmanD SDS | lld:pubmed |
| pubmed-article:1403646 | pubmed:author | pubmed-author:CollinsM KMK | lld:pubmed |
| pubmed-article:1403646 | pubmed:author | pubmed-author:JingS LSL | lld:pubmed |
| pubmed-article:1403646 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1403646 | pubmed:volume | 5 | lld:pubmed |
| pubmed-article:1403646 | pubmed:geneSymbol | tat | lld:pubmed |
| pubmed-article:1403646 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1403646 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1403646 | pubmed:pagination | 1142-7 | lld:pubmed |
| pubmed-article:1403646 | pubmed:dateRevised | 2008-11-21 | lld:pubmed |
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| pubmed-article:1403646 | pubmed:year | 1992 | lld:pubmed |
| pubmed-article:1403646 | pubmed:articleTitle | The human immunodeficiency virus tat protein increases the transcription of human Alu repeated sequences by increasing the activity of the cellular transcription factor TFIIIC. | lld:pubmed |
| pubmed-article:1403646 | pubmed:affiliation | Department of Biochemistry, University College and Middlesex School of Medicine, London, U.K. | lld:pubmed |
| pubmed-article:1403646 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1403646 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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