| pubmed-article:1341079 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:1341079 | lifeskim:mentions | umls-concept:C0035820 | lld:lifeskim |
| pubmed-article:1341079 | lifeskim:mentions | umls-concept:C0006826 | lld:lifeskim |
| pubmed-article:1341079 | lifeskim:mentions | umls-concept:C0007095 | lld:lifeskim |
| pubmed-article:1341079 | lifeskim:mentions | umls-concept:C0085695 | lld:lifeskim |
| pubmed-article:1341079 | lifeskim:mentions | umls-concept:C1326912 | lld:lifeskim |
| pubmed-article:1341079 | lifeskim:mentions | umls-concept:C0332281 | lld:lifeskim |
| pubmed-article:1341079 | lifeskim:mentions | umls-concept:C0449774 | lld:lifeskim |
| pubmed-article:1341079 | pubmed:issue | 6 | lld:pubmed |
| pubmed-article:1341079 | pubmed:dateCreated | 1994-1-6 | lld:pubmed |
| pubmed-article:1341079 | pubmed:abstractText | A series of 60 gastric endocrine tumors comprised 44 body-fundus argyrophil carcinoids, of which 23 arose in a background of hypergastrinemia and type A chronic atrophic gastritis (A-CAG), mainly with histologic patterns suggestive of an autoimmune process. Only 22 percent (compared with 19 percent of 58 tumor-free A-CAG cases) of 36 carcinoids and 21 percent of 19 A-CAG carcinoids investigated had Helicobacter pylori (HP) colonization, against 50 percent of 14 CAG-associated neuroendocrine carcinomas or mixed endocrine-exocrine tumors, 84 percent of 150 cases with early gastric cancer (p < 0.001 versus carcinoids), mostly with B- or AB-type CAG, 76 percent of 97 tumor-free AB-CAG, and 95 percent of 151 tumor-free B-CAG cases. Secondary hypergastrinemia and local mechanisms activated by chronic autoimmune gastritis are among factors involved in the pathogenesis of relatively indolent CAG-associated carcinoids, whereas active HP gastritis in cooperation with environmental carcinogens may likely cause more severe epithelial transformation, leading to ordinary cancer and, possibly, to neuroendocrine carcinomas or mixed endocrine-exocrine tumors. | lld:pubmed |
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| pubmed-article:1341079 | pubmed:language | eng | lld:pubmed |
| pubmed-article:1341079 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
| pubmed-article:1341079 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:1341079 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:1341079 | pubmed:issn | 0044-0086 | lld:pubmed |
| pubmed-article:1341079 | pubmed:author | pubmed-author:SolciaEE | lld:pubmed |
| pubmed-article:1341079 | pubmed:author | pubmed-author:CapellaCC | lld:pubmed |
| pubmed-article:1341079 | pubmed:author | pubmed-author:BuffaRR | lld:pubmed |
| pubmed-article:1341079 | pubmed:author | pubmed-author:RindiGG | lld:pubmed |
| pubmed-article:1341079 | pubmed:author | pubmed-author:VillaniLL | lld:pubmed |
| pubmed-article:1341079 | pubmed:author | pubmed-author:FioccaRR | lld:pubmed |
| pubmed-article:1341079 | pubmed:author | pubmed-author:AmbrosianiLL | lld:pubmed |
| pubmed-article:1341079 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:1341079 | pubmed:volume | 65 | lld:pubmed |
| pubmed-article:1341079 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:1341079 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:1341079 | pubmed:pagination | 793-804; discussion 827-9 | lld:pubmed |
| pubmed-article:1341079 | pubmed:dateRevised | 2010-9-7 | lld:pubmed |
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| pubmed-article:1341079 | pubmed:articleTitle | Distinct patterns of chronic gastritis associated with carcinoid and cancer and their role in tumorigenesis. | lld:pubmed |
| pubmed-article:1341079 | pubmed:affiliation | Department of Human Pathology and Genetics, I Medical Faculty, University of Pavia, Italy. | lld:pubmed |
| pubmed-article:1341079 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:1341079 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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