Linked Life Data

12837073

Source:http://linkedlifedata.com/resource/pubmed/id/12837073

Statements in which the resource exists as a subject.
PredicateObject
rdf:type
lifeskim:mentions
pubmed:issue
27
pubmed:dateCreated
2003-7-2
pubmed:abstractText
Computational evidence at the CASPT2 level supports that the lowest excited state pipi* contributes to the S1/S0 crossing responsible for the ultrafast decay of singlet excited cytosine. The computed radiative lifetime, 33 ns, is consistent with the experimentally derived value, 40 ns. The nOpi* state does not play a direct role in the rapid repopulation of the ground state; it is involved in a S2/S1 crossing. Alternative mechanisms through excited states pisigma* or nNpi* are not competitive in cytosine.
pubmed:language
eng
pubmed:journal
pubmed:citationSubset
IM
pubmed:chemical
pubmed:status
MEDLINE
pubmed:month
Jul
pubmed:issn
0002-7863
pubmed:author
pubmed:issnType
Print
pubmed:day
9
pubmed:volume
125
pubmed:owner
NLM
pubmed:authorsComplete
Y
pubmed:pagination
8108-9
pubmed:dateRevised
2008-1-17
pubmed:meshHeading
pubmed:year
2003
pubmed:articleTitle
Ultrafast internal conversion of excited cytosine via the lowest pipi electronic singlet state.
pubmed:affiliation
Departamento de Química Física, Instituto de Ciencia Molecular, Universitat de València, Dr. Moliner 50, Burjassot, ES-46100 Valencia, Spain. manuela.merchan@uv.es
pubmed:publicationType
Journal Article, Research Support, Non-U.S. Gov't