pubmed-article:12773140 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:12773140 | lifeskim:mentions | umls-concept:C0086418 | lld:lifeskim |
pubmed-article:12773140 | lifeskim:mentions | umls-concept:C0299583 | lld:lifeskim |
pubmed-article:12773140 | lifeskim:mentions | umls-concept:C0229671 | lld:lifeskim |
pubmed-article:12773140 | lifeskim:mentions | umls-concept:C0019134 | lld:lifeskim |
pubmed-article:12773140 | lifeskim:mentions | umls-concept:C0574032 | lld:lifeskim |
pubmed-article:12773140 | lifeskim:mentions | umls-concept:C0733854 | lld:lifeskim |
pubmed-article:12773140 | pubmed:issue | 6 | lld:pubmed |
pubmed-article:12773140 | pubmed:dateCreated | 2003-5-29 | lld:pubmed |
pubmed-article:12773140 | pubmed:abstractText | Our previous studies showed that administration of dexamethasone plus food increased serum leptin levels 100% more than dexamethasone alone. We hypothesized that this increase in leptin from the meal could result directly from the provision of fuel metabolites rather than from the meal-induced rise in insulin. In the current study, we tested the effect of an i.v. lipid fuel source (Intralipid 20%/heparin) that would incur only a modest increase in insulin. This study was undertaken because the role of lipid in the regulation of human leptin levels has been controversial, with differing effects reported: stimulatory, inhibitory, or no effect at all. | lld:pubmed |
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pubmed-article:12773140 | pubmed:language | eng | lld:pubmed |
pubmed-article:12773140 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:12773140 | pubmed:citationSubset | IM | lld:pubmed |
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pubmed-article:12773140 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:12773140 | pubmed:month | Jun | lld:pubmed |
pubmed-article:12773140 | pubmed:issn | 0804-4643 | lld:pubmed |
pubmed-article:12773140 | pubmed:author | pubmed-author:LaferrereBB | lld:pubmed |
pubmed-article:12773140 | pubmed:author | pubmed-author:Garcia-LordaP... | lld:pubmed |
pubmed-article:12773140 | pubmed:author | pubmed-author:NashWendyW | lld:pubmed |
pubmed-article:12773140 | pubmed:author | pubmed-author:RocheAnsleyA | lld:pubmed |
pubmed-article:12773140 | pubmed:author | pubmed-author:Pi-SunyerF-XF... | lld:pubmed |
pubmed-article:12773140 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:12773140 | pubmed:volume | 148 | lld:pubmed |
pubmed-article:12773140 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:12773140 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:12773140 | pubmed:pagination | 669-76 | lld:pubmed |
pubmed-article:12773140 | pubmed:dateRevised | 2011-11-17 | lld:pubmed |
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pubmed-article:12773140 | pubmed:meshHeading | pubmed-meshheading:12773140... | lld:pubmed |
pubmed-article:12773140 | pubmed:meshHeading | pubmed-meshheading:12773140... | lld:pubmed |
pubmed-article:12773140 | pubmed:year | 2003 | lld:pubmed |
pubmed-article:12773140 | pubmed:articleTitle | Intralipid/heparin infusion suppresses serum leptin in humans. | lld:pubmed |
pubmed-article:12773140 | pubmed:affiliation | Obesity Research Center, St Luke's/Roosevelt Hospital Center, Columbia University College of Physicians and Surgeons, 1111 Amsterdam Avenue, New York, NY 10025, USA. | lld:pubmed |
pubmed-article:12773140 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:12773140 | pubmed:publicationType | Clinical Trial | lld:pubmed |
pubmed-article:12773140 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
pubmed-article:12773140 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
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