| pubmed-article:12019235 | rdf:type | pubmed:Citation | lld:pubmed |
| pubmed-article:12019235 | lifeskim:mentions | umls-concept:C0282641 | lld:lifeskim |
| pubmed-article:12019235 | lifeskim:mentions | umls-concept:C0687129 | lld:lifeskim |
| pubmed-article:12019235 | lifeskim:mentions | umls-concept:C1152517 | lld:lifeskim |
| pubmed-article:12019235 | lifeskim:mentions | umls-concept:C0013139 | lld:lifeskim |
| pubmed-article:12019235 | lifeskim:mentions | umls-concept:C0851285 | lld:lifeskim |
| pubmed-article:12019235 | lifeskim:mentions | umls-concept:C0013879 | lld:lifeskim |
| pubmed-article:12019235 | lifeskim:mentions | umls-concept:C2700400 | lld:lifeskim |
| pubmed-article:12019235 | pubmed:issue | 1 | lld:pubmed |
| pubmed-article:12019235 | pubmed:dateCreated | 2002-5-20 | lld:pubmed |
| pubmed-article:12019235 | pubmed:abstractText | Fusions between the Drosophila hsp70 promoter and three different incomplete P elements, KP, SP, and BP1, were inserted into the Drosophila genome by means of hobo transformation vectors and the resulting transgenic stocks were tested for repression of P-element transposase activity. Only the H(hsp/KP) transgenes repressed transposase activity, and the degree of repression was comparable to that of a naturally occurring KP element. The KP transgenes repressed transposase activity both with and without heat-shock treatments. Both the KP element and H(hsp/KP) transgenes repressed the transposase activity encoded by the modified P element in the P(ry(+), Delta2-3)99B transgene more effectively than that encoded by the complete P element in the H(hsp/CP)2 transgene even though the P(ry(+), Delta2-3)99B transgene was the stronger transposase source. Repression of both transposase sources appeared to be due to a zygotic effect of the KP element or transgene. There was no evidence for repression by a strictly maternal effect; nor was there any evidence for enhancement of KP repression by the joint maternal transmission of H(hsp/KP) and H(hsp/CP) transgenes. These results are consistent with the idea that KP-mediated repression of P-element activity involves a KP-repressor polypeptide that is not maternally transmitted and that KP-mediated repression is not strengthened by the 66-kD repressor produced by complete P elements through alternate splicing of their RNA. | lld:pubmed |
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| pubmed-article:12019235 | pubmed:language | eng | lld:pubmed |
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| pubmed-article:12019235 | pubmed:citationSubset | IM | lld:pubmed |
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| pubmed-article:12019235 | pubmed:status | MEDLINE | lld:pubmed |
| pubmed-article:12019235 | pubmed:month | May | lld:pubmed |
| pubmed-article:12019235 | pubmed:issn | 0016-6731 | lld:pubmed |
| pubmed-article:12019235 | pubmed:author | pubmed-author:SimmonsMichae... | lld:pubmed |
| pubmed-article:12019235 | pubmed:author | pubmed-author:HaleyKevin... | lld:pubmed |
| pubmed-article:12019235 | pubmed:author | pubmed-author:GrimesCraig... | lld:pubmed |
| pubmed-article:12019235 | pubmed:author | pubmed-author:RaymondJohn... | lld:pubmed |
| pubmed-article:12019235 | pubmed:author | pubmed-author:FongJoseph... | lld:pubmed |
| pubmed-article:12019235 | pubmed:issnType | Print | lld:pubmed |
| pubmed-article:12019235 | pubmed:volume | 161 | lld:pubmed |
| pubmed-article:12019235 | pubmed:owner | NLM | lld:pubmed |
| pubmed-article:12019235 | pubmed:authorsComplete | Y | lld:pubmed |
| pubmed-article:12019235 | pubmed:pagination | 205-15 | lld:pubmed |
| pubmed-article:12019235 | pubmed:dateRevised | 2010-9-14 | lld:pubmed |
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| pubmed-article:12019235 | pubmed:meshHeading | pubmed-meshheading:12019235... | lld:pubmed |
| pubmed-article:12019235 | pubmed:year | 2002 | lld:pubmed |
| pubmed-article:12019235 | pubmed:articleTitle | Regulation of P-element transposase activity in Drosophila melanogaster by hobo transgenes that contain KP elements. | lld:pubmed |
| pubmed-article:12019235 | pubmed:affiliation | Department of Genetics, Cell Biology and Development, University of Minnesota, Saint Paul, Minnesota 55108-1095, USA. simmo004@tc.umn.edu | lld:pubmed |
| pubmed-article:12019235 | pubmed:publicationType | Journal Article | lld:pubmed |
| pubmed-article:12019235 | pubmed:publicationType | Research Support, U.S. Gov't, P.H.S. | lld:pubmed |
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