pubmed-article:11849426 | rdf:type | pubmed:Citation | lld:pubmed |
pubmed-article:11849426 | lifeskim:mentions | umls-concept:C0028128 | lld:lifeskim |
pubmed-article:11849426 | lifeskim:mentions | umls-concept:C0162638 | lld:lifeskim |
pubmed-article:11849426 | lifeskim:mentions | umls-concept:C0227651 | lld:lifeskim |
pubmed-article:11849426 | pubmed:issue | 3 | lld:pubmed |
pubmed-article:11849426 | pubmed:dateCreated | 2002-2-18 | lld:pubmed |
pubmed-article:11849426 | pubmed:abstractText | Nitric oxide and apoptosis in mesangial cells. Radicals such as nitric oxide (NO) or superoxide (O(-)(2)) encompass the ability to initiate morphological and biochemical alterations that are reminiscent of apoptosis. In mesangial cells, death as a result of NO formation is efficiently antagonized by the simultaneous presence of superoxide (O(-)(2)) and vice versa. This article reviews the consequences of a diffusion controlled NO/O(-)(2) interaction with the outcome of redirecting the apoptotic initiating activity of either NO or O(-)(2) toward protection. The crosstalk between cell destructive and protective pathways, and their activation or inhibition under the modulatory influence of NO and/or O(-)(2) are summarized. | lld:pubmed |
pubmed-article:11849426 | pubmed:language | eng | lld:pubmed |
pubmed-article:11849426 | pubmed:journal | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11849426 | pubmed:citationSubset | IM | lld:pubmed |
pubmed-article:11849426 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11849426 | pubmed:chemical | http://linkedlifedata.com/r... | lld:pubmed |
pubmed-article:11849426 | pubmed:status | MEDLINE | lld:pubmed |
pubmed-article:11849426 | pubmed:month | Mar | lld:pubmed |
pubmed-article:11849426 | pubmed:issn | 0085-2538 | lld:pubmed |
pubmed-article:11849426 | pubmed:author | pubmed-author:BrüneBernhard... | lld:pubmed |
pubmed-article:11849426 | pubmed:issnType | Print | lld:pubmed |
pubmed-article:11849426 | pubmed:volume | 61 | lld:pubmed |
pubmed-article:11849426 | pubmed:owner | NLM | lld:pubmed |
pubmed-article:11849426 | pubmed:authorsComplete | Y | lld:pubmed |
pubmed-article:11849426 | pubmed:pagination | 786-9 | lld:pubmed |
pubmed-article:11849426 | pubmed:dateRevised | 2006-11-15 | lld:pubmed |
pubmed-article:11849426 | pubmed:meshHeading | pubmed-meshheading:11849426... | lld:pubmed |
pubmed-article:11849426 | pubmed:meshHeading | pubmed-meshheading:11849426... | lld:pubmed |
pubmed-article:11849426 | pubmed:meshHeading | pubmed-meshheading:11849426... | lld:pubmed |
pubmed-article:11849426 | pubmed:meshHeading | pubmed-meshheading:11849426... | lld:pubmed |
pubmed-article:11849426 | pubmed:meshHeading | pubmed-meshheading:11849426... | lld:pubmed |
pubmed-article:11849426 | pubmed:meshHeading | pubmed-meshheading:11849426... | lld:pubmed |
pubmed-article:11849426 | pubmed:year | 2002 | lld:pubmed |
pubmed-article:11849426 | pubmed:articleTitle | Nitric oxide and apoptosis in mesangial cells. | lld:pubmed |
pubmed-article:11849426 | pubmed:affiliation | Department of Cell Biology, University of Kaiserslautern, Erwin-Schrödinger-Strasse, 67663 Kaiserslautern, Germany. bruene@rhrk.uni-kl.de | lld:pubmed |
pubmed-article:11849426 | pubmed:publicationType | Journal Article | lld:pubmed |
pubmed-article:11849426 | pubmed:publicationType | Review | lld:pubmed |
pubmed-article:11849426 | pubmed:publicationType | Research Support, Non-U.S. Gov't | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11849426 | lld:pubmed |
http://linkedlifedata.com/r... | pubmed:referesTo | pubmed-article:11849426 | lld:pubmed |