Source:http://linkedlifedata.com/resource/pubmed/id/11721751
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Predicate | Object |
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rdf:type | |
lifeskim:mentions | |
pubmed:issue |
11
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pubmed:dateCreated |
2001-11-27
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pubmed:abstractText |
The development of reflux esophagitis in humans is a process resulting from esophageal exposure to refluxed gastric contents. There is no doubt that damage to the esophageal epithelium requires exposure to gastric acid; however, the role of refluxed pepsin as contributor to this damage seems to be underappreciated.
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pubmed:grant | |
pubmed:language |
eng
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pubmed:journal | |
pubmed:citationSubset |
IM
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pubmed:chemical | |
pubmed:status |
MEDLINE
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pubmed:month |
Nov
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pubmed:issn |
0002-9270
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pubmed:author | |
pubmed:issnType |
Print
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pubmed:volume |
96
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pubmed:owner |
NLM
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pubmed:authorsComplete |
Y
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pubmed:pagination |
3062-70
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pubmed:dateRevised |
2007-11-14
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pubmed:meshHeading | |
pubmed:year |
2001
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pubmed:articleTitle |
The role of pepsin in acid injury to esophageal epithelium.
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pubmed:affiliation |
Department of Medicine, Tulane University School of Medicine and the Veterans Administration Medical Center, New Orleans, Louisiana 70112, USA.
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pubmed:publicationType |
Journal Article,
Research Support, U.S. Gov't, P.H.S.
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