rdf:type |
|
lifeskim:mentions |
|
pubmed:issue |
8
|
pubmed:dateCreated |
2001-10-30
|
pubmed:abstractText |
Pulmonary edema, owing to an impairment of microvascular barrier function, is an important feature in lung ischemia/reperfusion (IR) injury. Inhalation of nitric oxide (NO) during the period of reperfusion has previously been shown to reduce this leakage response.
|
pubmed:language |
eng
|
pubmed:journal |
|
pubmed:citationSubset |
IM
|
pubmed:chemical |
|
pubmed:status |
MEDLINE
|
pubmed:month |
Oct
|
pubmed:issn |
0041-1337
|
pubmed:author |
|
pubmed:issnType |
Print
|
pubmed:day |
27
|
pubmed:volume |
72
|
pubmed:owner |
NLM
|
pubmed:authorsComplete |
Y
|
pubmed:pagination |
1363-70
|
pubmed:dateRevised |
2006-11-15
|
pubmed:meshHeading |
pubmed-meshheading:11685104-Administration, Inhalation,
pubmed-meshheading:11685104-Animals,
pubmed-meshheading:11685104-Cyclic GMP,
pubmed-meshheading:11685104-Female,
pubmed-meshheading:11685104-Ischemic Preconditioning,
pubmed-meshheading:11685104-Lung,
pubmed-meshheading:11685104-Male,
pubmed-meshheading:11685104-Nitric Oxide,
pubmed-meshheading:11685104-Pulmonary Artery,
pubmed-meshheading:11685104-Rabbits,
pubmed-meshheading:11685104-Reperfusion Injury,
pubmed-meshheading:11685104-Weight Gain
|
pubmed:year |
2001
|
pubmed:articleTitle |
Short-term "preconditioning" with inhaled nitric oxide protects rabbit lungs against ischemia-reperfusion injury.
|
pubmed:affiliation |
Department of Internal Medicine, Justus-Liebig University, Klinikstrasse 36, 35385 Giessen, Germany. hartwig.schuette@innere.med.uni-giessen.de
|
pubmed:publicationType |
Journal Article,
Research Support, Non-U.S. Gov't
|